To solve the argument about the relationship between arcuate fasciculus and conduction aphasia, more research into the brain activity of individuals with and without symptoms of conduction aphasia should be conducted, so that we could have a clearer understanding about the mechanism of how different areas of the brain are related to speech repetition problems. In fact, studies using FMRI have found that certain population have Broca’s and Wernicke’s located in different hemisphere of their brain (Bernal & Ardila, 2009). For these subjects, their Broca’s and Wernicke’s areas are inherently dissociated. However, no problems in speech repetition have been reported in these individuals. In addition, Kempler and his colleagues (1988) used FMRI to …show more content…
Bernal and Ardila (2009) speculated that arcuate fasciculus is an accessory aid in transmitting information from the temporal lobe to premotor or motor cortex, but not directly to Broca’s area. According to Bernal and Ardila (2009), conduction aphasia is more likely to be a consequence resulting from premotor disconnection, which could impair the motor ability required for word sequencing and producing word syllables or segments in a proper order. Given that language areas in the brain are not damaged, patients with conduction aphasia can still retain their phonological awareness, and this is the reason why they can still be aware of their speech error and attempt to correct them. By contrast, for patients suffering from phonological paraphasia, they are not able to detect their own errors since the language areas in their brain are affected. In this model suggested by Bernal and Ardila (2009), arcuate fasciculus serves as a relay station to connect the Broca’s area with the premotor or motor areas. It clearly explains why arcuate fasciculus is closer to speech (motor function) rather than to language, and is also more congruent with the current neuroimaging results and clinical findings (Bernal & Ardila,
Wernicke's Aphasia occurs from damage to the superior temporal gyrus and affects language comprehension, while speech production is unaffected, which is why it is referred to as "fluent aphasia." Wernicke's patients produce speech fluently, but it is mostly nonsensical speech and have trouble understanding what is being told to them. Unlike Broca's Aphasia patients, they are not aware of their speech problems.
Seikel, J. A., King, D. W., & Drumright, D. G. (2010). 12. Anatomy & physiology for speech,
The temporal lobe of the cerebral cortex is associated with the processing of language. During verbal learning tests on subjects who are fully rested functional magnetic resonance imaging scans show that this area of the brain is very active. However, in sleep deprived subjects there is no activity within this region (3), (4), (5). The effects of this inactivity can be observed by the slurred speech in subjects who have gone for prolonged periods with no sleep (6).
Aphasia can be defined as a disorder that is caused by damage to parts of the brain that are responsible for language (“Aphasia” n.p.). Wernicke’s aphasia is a type of fluent aphasia (with the other type being nonfluent). It is named after Carl Wernicke who described the disorder as “an amnesiac disorder characterized by fluent but disordered speech, with a similar disorder in writing, and impaired understanding of oral speech and reading” (“Wernicke’s” n.p.). Wernicke’s aphasia can also be known as sensory aphasia, fluent aphasia, or receptive aphasia. It is a type of aphasia that is caused by damage to Wernicke’s area in the brain, in the posterior part of the temporal lobe of the left hemisphere. This area of the brain contains motor neurons responsible for the understanding of spoken language and is believed to be the receptive language center (“Rogers” n.p.). Wernicke’s aphasia can be most efficiently defined as a fluent language disorder commonly caused by strokes and characterized by difficulty comprehending spoken language and producing meaningful speech and writing which is both assessable by an SLP and treatable by a variety of methods.
Kanske, P., Heissler, J., Schönfelder, S., Forneck, J., & Wessa, M. (2013). Neural correlates of
...tion. In true recognition, there was more activity in temporal lobe on left hemisphere, which store sounds of words.
Caramazza, A., & Coltheart, M. (2006). Cognitive Neuropsychology twenty years on. Cognitive Neuropsychology, Vol. 23, pp. 3-12.
The Phonological Deficit and Magnocellular theory are two of the most dominant theories in dyslexic research. Various theories have been suggested to explain the nature and origin of dyslexia, however, they often served as additional support for either the phonological or magnocellular theories. The Double Deficit theory suggested that dyslexic symptoms were the result of speed-processing (7). The Genomic theory posed that dyslexia was a highly heritable disorder that can be localized to a specific genetic component, Finally, the Cerebellar Deficit theory suggested that dyslexia was the result of an abnormal cerebellum exist (2). With the constant debate of the biological nature versus the cognitive natur...
Speech language pathology is another major rehabilitative medical care. Some stroke survivors area unit left with brain disorder, an impairment of language and speaking skills within which the stroke survivor will assume likewise as before the stroke, however is unable to induce the proper words out or is unable to method words coming back [32]. Brain disorder is sometimes caused by a stroke on the left aspect of the brain. Speech language pathology will teach the aphasic stroke survivor and his or her family members’ ways for dealing with this frustrating impairment. Speech language pathologists additionally work to assist the stroke survivor deal with blackout and alternative "thought" issues caused by the stroke [33].
Researchers have provided different classifications of speech errors. They can be categorized according to the “linguistic units,” such as “phonological feature, phoneme, syllable, morpheme, word phrase, or sentence levels” (Harely, 2001, p. 376). Moreover, speech errors can be classified according to the “mechanisms” of the speech errors (Harely, 2001, p. 376). For example, Carroll (2007) classified eight of the basic types of slips of the tongue according to the error mechanism from the previous psycholinguistic studies. These errors include shift, exchanges, anticipations, perseveration, additions, deletions, substitutions, and blends.
In physiological terms, Broca's aphasia and Wernicke's aphasia occur in the left hemisphere of the brain, which is responsible for controlling the right side of the body along with speech and language abilities. Broca's aphasia affects the frontal lobe adjacent to the primary motor cortex, and Wernicke's aphasia affects the posterior portion of the first frontal lobe (3). A general distinction made between the two disorders are that Broca's aphasia limits speech, while Wernicke's aphasia limits comprehension.
Salonen, L. (2013). L. S. Vygotsky 's psychology and theory of learning applied to the rehabilitation of aphasia: A developmental and systemic view. Aphasiology, 27(5), 615-635. doi:10.
The causes of stuttering are not very well understood. There is some evidence that ...
Frontal Lobe Syndrome Although volumetrically the frontal lobes are the largest portion of the brain their function remains somewhat elusive (Jacobs, 2005). Even neuropsychologists have a difficult time creating test that accurately test frontal lobe functioning. We do know however, that the frontal lobes are involved in the storage of memories, concentration, abstract thought, judgment, and self control. The frontal lobe lies directly behind our forehead (NINDS, 2005)
In this world, humans and animals alike have come to communicate by using various mechanisms. Humans have advanced themselves beyond other organisms by using language, or a set of codes and symbols, in order to express themselves to others. Language has brought about a means to create new thoughts, to explore, and to analyze our everyday surroundings. It has also enabled us to retain past memories and to look deep into the advances for the future. However, for some individuals, this tool for communication has been plagued by a language and speech disorders, such as aphasia. Aphasia is the loss of the ability to speak or understand speech or written language. It is often detected at an early age, and contributes to the general class of speech and language disorders affecting "5% of school aged children" (1) . Aphasia is classified into three categories. The main two are receptive or sensory aphasia and expressive or motor aphasia. Receptive aphasia affects the input side and "the ability to understand spoken or written language may be partially or totally lost" (1) . Those with expressive aphasia "can speak but not find certain words or names, or may be totally unable to communicate verbally or by writing" (1) . For a majority of affected individuals, there is a combination of the two. The third type is conduction aphasia. This "involves disruption of transmission between the sensory and motor ends of the circuit" (1) . Here, individuals are able to produce speech despite the lack of connections to the input side. It seems that the ability to speak has a lot to do with your surroundings and how much emphasis was placed on developing this skill during the first few years after birth. Afterall, it's known that the first few years are critical because this is the time when the brain is "plastic" and is rapidly changing and being molded. By the time that adolescence is reached, the brain has become "less plastic". In this paper, I would like to explore theories proposed to try to understand the origins of this impairment.