Anatomy and Physiology of Diabetic Retinopathy

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Anatomy and Physiology of Diabetic Retinopathy

Retinopathy is usually found in people that have high blood pressure and diabetics. It is most commonly in people that have diabetes. The cause of retinopathy occurs when the blood vessels swell and leak fluid or even close off completely. Some cases abnormal new blood cells grows on the surface of the retina. People with diabetic retinopathy get it in both eyes. Most diabetics do not notice any visual complications at an early stage’s of the disease. As this disease progresses, usually cause vision loss that usually cannot be reversed. Different parts of the retina can be affected like the macula, the outer part of the retina or both. Which can make retinopathy more serious. Background retinopathy doesn’t change the sight. With this you have tiny leaks fluid and tiny bleeds in various parts of the retina. Pre-proliferative retina is more extensive, signs of blood flow becoming more restive, but not yet showing signs of new blood vessels growing. Maculopathy shows damage to the macula. Proliferative retinopathy when damage blood vessels in the retina make chemicals called growth factors. The form new blood vessels which are not normal. Without laser treatment, vision is most likely become badly affected.

The cornea is the dome shape outer covering of the eye and also where light is focused. There are no blood vessels in the cornea due to the blood vessels would block the light from entering. It receive it nutrients and oxygen from tears and aqueous humor. The sclera is the outer white portion of the eye. Which supply protection and structure of the inner parts of the eye. Conjunctiva is a mucus layer that keep the eyes moist. This also covers the sclera and the ...

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...s leads to insufficient transportation of glucose into the cells for energy production and conversion of excess glucose into glycogen or fat storage. So glucose accumulates in the blood plasma causing hyperglycemia.

References

Works Cited

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