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    results gained from accurate experimental procedures. Aim The aim of this experiment is to investigate the rate at which iodine is formed when the concentration and temperature of the reactants are varied, and to attempt to find the order and activation energy. The Chemistry 'THE IODINE CLOCK' - This is the experiment that will be used to investigate reaction rates, and it is a reaction between acidified hydrogen peroxide and potassium iodide: 2H+(aq) + 2I¯ (aq) + H[-1] 2O2 (l) ÕI2 (aq)

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    temperature of the solution in which the reaction is taking place. From these results, I hope to draw conclusions as to the effects of these changes to the environment of the reaction on the rate and also determine the order of the reaction and the activation enthalpy. Background information The rate of a reaction is determined by a number of factors. These include: pressure, temperature, concentration of reactants, surface area of reactants, presence of a catalyst and radiation. The effect of these

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    Proposed Mechanisms of Dreaming

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    behaviors accompanying REM sleep (8). These behaviors or characteristics include: -phasic clusters of extraocular muscles of the eye producing rapid eye movement -generalized activation of the forebrain (cerebral cortex) -phasic activation of the visual pathway -inhibition of sensory input -suppressed motor activity -activation or inhibition of various brain stem neurons. (5) Many of these behavioral markers associated with REM sleep closely dictate or reflect the dream phenomenon. Although the

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    Theoretical Approaches to Speech Production There are two main theories of Speech production, Spreading Activation Theory - SAT (Dell, 1986: Dell & O’Seaghdha, 1991) and Word- Form Encoding by Activation and Verification – WEAVER++ (Levelt et al., 1989: 1999). The SAT theory was devised by Dell (1986) then revised by Dell & O’Seaghda (1991). The theory works on a 4 level connectionist model: parallel and dynamic. The Semantic level is the meaning of what is going to be said. The Syntactic

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    and serotonin, which helped to develop a greater understanding of the role serotonin plays as a neurotransmitter in the brain (Nichols, 2004). Today it is believed that LSD (and other hallucinogens) stimulate 5-HT2A receptors (Kalat, 2004). Activation of these receptors causes cortical glutamate levels to increase. This is presumed to be a result of a "presynaptic receptor-mediated release" from neurons in the thalumus (Nichols, 2004). Early studies proposed that LSD antagonized the effects

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    out of military compounds that were equipped with all types of security systems. Somehow he would always find the laser beams, or trick the fingerprint security, or voice activation. During that time biometrics was just beginning to come to the surface. Nowadays systems enabled with fingerprinting security, voice activation, or face recognition have come a long way and are close to fool proof. Biometrics have come even as far to be used at the Super Bowl or other huge sporting events. Cameras

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    reaction. This is due to the energy barrier to overcome. Only particles with enough energy to overcome the barrier will react after colliding. The minimum energy that a particle must have to overcome the barrier is called the activation energy, or Ea. The size of this activation energy is different for different reactions. If the frequency of collisions is increased the rate of reaction will increase. However the percent of successful collisions remains the same. An increase in the frequency of

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    Fear of Fear

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    restructuring aimed at teaching patients to identify and correct faulty threat perceptions that contribute to their panic and anxiety, interoceptive exposure aimed at reducing patents’ fear of harmless bodily sensations associated with physiological activation, and fading of maladaptive defensive behaviors such as avoidance of external situations (Barlow, Craske, Cerny, & Klosko, 1989; Clark et al., 1994; Telch et al., 199). On the basis of contemporary psychological theories of panic disorder, several

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    can see that pain is a perception, sort of like seeing and hearing. When pain is processed there are a number of brain structures activated, commonly referred to as the "central pain matrix" (2). It may seem irrelevant to delve into pain signal activation in the brain since it is seemi... ... middle of paper ... ...that one's entire perception of pain may be conscious in origin and simply correlate to the mechanisms of the brain, rather than stem from just the brain entirely. References

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    numerous intracytoplasmic granules and are the source of numerous proinflammatory mediators. In fact, they are quantitatively the greatest single source of vasoactive amines in the body. They also are a rich source of thromboxane A2. It is their activation that, in part, initiates the vascular phase of the acute inflammatory response (see Fig. 2-13 in text). To have them play this role makes imminently good sense, because they are present in large numbers throughout the circulation, i.e., some are

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