Caffeine Report

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Improvement of aerobic exercise after acute caffeine consumption
Introduction
Caffeine as a member of the methylxanthine class, is a central nervous system stimulant which can suppress the effect of adenosine on adenosine receptors in nerve tissues and consequently reduces the drowsiness induced by adenosine. (Nehlig et al., 1992) Caffeine is the most commonly used psychoactive substance as unlike most methylxanthines, not only can it be legally obtained, it also remains unregulated. Current studies have shown that low-to-moderate dosages of caffeine can improve both cognitive performance and physical performance. At low dosage, it can effectively stimulate higher attention and by reducing anxiety and fatigue, caffeine can facilitate on short-term …show more content…

(Nehlig, 2010) It is also found that moderate dosages of caffeine can enhance trained athletes’ sport performance by improving vigilance and benefits high-intensity exercise. (Goldstein et al., 2010) These notable effects make beverages containing caffeine including coffee, tea and energy drink popular and lead to the rapid growth in caffeine market targeting particularly to youth. However, the high accessibility also leads to the problem of overconsumption of caffeine and increasing reports of caffeine toxicity from energy drinks particularly among adolescents. (Gunja and Brown, 2011). Researches also showed that caffeine abuse causes insomnia and large doses of caffeine will show reverse symptoms as consumption of low dosages including agitation and recurrent headache and gastrointestinal disturbance. (Bolton, 1981) Therefore, more researches are required to study the functioning of caffeine in human bodies to …show more content…

The pre-treatment resting pulse rate was expected to be same as healthy people who were not trained athletes should have similar pulse rate ranging from 60 beats per minute to 100 beats per minute. In the caffeinated treatment group, the resting pulse rate increases after acute consumption of caffeine which corresponded with the prediction of caffeine’s effect in increasing heart rate, but the increase is not substantial, thus it may be due to errors. However, no significance in difference in post-treatment resting pulse rate and post-exercise pulse rate indicated that caffeine did not contribute to the increase in pulse rate during exercise. Thus, the result rejected the hypothesis of caffeine’s effect of increasing pulse rate/aerobic capacity during

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