Peripheral neuropathy is a term which describes damage to one or more of your peripheral nerves. The damage means that the messages that travel between your central and peripheral nervous system are disrupted. There are many different conditions that can lead to peripheral neuropathy. Diabetes is known as the major cause of persistent peripheral neuropathy. The symptoms and the cause of peripheral neuropathy depend on which types of peripheral nerves are damaged. Neuropathy can affect any one, both the young and the old. However, people suffering from diabetes and other infections have a high possibility of being affected with this disease. The aims of treatment for peripheral neuropathy are to treat any underlying cause, to control your symptoms and to help you to achieve maximum independence. There are more than a hundred types of peripheral neuropathy disorder that has been identified in different patients, each revealing different kind of symptoms from the other as well as pattern of development. Impaired function of nerves depend on the kind of damaged that the immune is suffering from. Some victims may experience numbness, tingling, and pricking sensations, sensitive touch and weakness of the muscles. What causes this disease? Peripheral neuropathy can be attracted in two different ways, either through inheritance or caused by other infection. Other disorders that can cause this disease include tumors, excessive toxins in the body, negative responses caused by the immune system, nutritional and vitamin deficiencies, excessive consumption of alcohol and metabolic disorders. Peripheral neuropathy is categorized into three groups. This classification is determined by the cause of the disease, which makes it easy for doctors ... ... middle of paper ... ... for this disorder depends on the cause of problem. It is also advisable for one to go for tests before applying any type of treatment. People diagnosed with diabetes are advised to seek treatment for any type of ailment that may interfere with the nervous system. This makes it easy to cure peripheral neuropathy since other infections will have been eradicated before treating this disorder. These tests are also conducted to conclude the right medication for patients, who are suffering from this nerve damage. Another factor that you should note is that, the kind of treatment being applied to victims also depends on the age of the victims since young adults and children cannot be diagnosed with the same medication. Some cases of peripheral neuropathy can be treated, while others cannot be treated especially if the victim inherited the disorder from a family member.
Each sensation has its own neuronal receptor, such as: “mechanosensation, thermosensation, vibration, joint position, chemosensation, and electrosensation.” Oaklander then discusses “nocifensive sensations,” or senses that defend us from danger, such as pain and itch. These sensations trigger reflexes and strong movements. However, something that is often left undetected is chronic neuropathic pain, which can cause nerve damage. Shingles is a result of chronic neuropathic pain.
MS causes a degeneration of the myelin around axons due to the killing off of oligodendrocytes, which are cells that make up the myelin sheath of an axon; losing myelin decreases the neuron’s ability to propagate an action potential. Since this disease affects the central nervous system, MS can cause dysfunction of both the sensory and the motor aspects of the body. Some common sensory complaints(Lundy-Ekman, 2007) of MS are tingling, numbness, and/or paresthesia in the affected area, which is variable but typically involves one or more limbs (Palace, 2001), as well as partial blindness in one eye, a decrease in vision acuity, and double vision. Lhermitte’s sign, which is a radiating shock that travels down the back or limbs, is another common characteristic of MS that aff...
In the United States 54 million people have a disability and only 15 percent were born with a disability (Jaeger & Bowman, 2005). If a person lives long enough, it is statistically likely that they will develop some kind of disability in their advancing years (Jaeger & Bowman, 2005). At some point in your life you could have experience a fractured bone, a minor cut, or had some type of surgery. Imagine after some minor injury that you may not even remember and then experiencing a constant pain so agonizing that no amount of pain medication can make you comfortable (Lang & Moskovitz, 2003). Some additional symptoms that you may also experience are severe burning pain, changes in bone and skin, excessive sweating, tissue swelling and extreme sensitivity to touch (Juris, 2005). These symptoms are associated with a disease that is called Reflex Sympathetic Dystrophy (RSD) but more recently termed as complex regional pain syndrome, type 1 (CRPS 1) (Juris, 2005). For simplification purposes this disease will be referred to as RSD throughout this paper.
PD is separated into stages according to the symptoms and degree of disability Stage 1 is mild disease with unilateral limb involvement. Whereas the patient with stage 5 disease is completely dependent in all ADLs. Other classification refer simply to mild, moderate, and ...
Parkinson's is an idiopathic, multifactorial neurodegenerative disease that attacks neurotransmitters in the brain called dopamine. Dopamine is concentrated in a specific area of the brain called the substantia nigra. The neurotransmitter dopamine is a chemical that regulates muscle movement and emotion. Dopamine is responsible for relaying messages between the substantia nigra and other parts of the brain to control body movement. The death of these neurotransmitters affects the central nervous system. The most common symptoms are movement related, including shaking, rigidity, slowness of movement and difficulty with posture. Behavioral problems may arise as the disease progresses. Due to the loss of dopamine, Parkinson's patients will often experience depression and some compulsive behavior. In advanced stages of the disease dementia will sometimes occur. The implications of the disease on the anatomy and physiology of the respiratory and phonatory systems significantly control speech.
Peripheral artery disease is usually caused by atherosclerosis, which is when fatty deposits accumulate in the arteries of, most likely, limbs. This does affect all arteries throughout the body, then in turn, slowing the blood flow. PAD may also be due to inflammation of the blood vessels, injury to the extremities, or even exposure to radiation. “Peripheral arterial disease (PAD) affects approximately 10% of the American population” (Gurbir Dhaliwal; Peripheral arterial disease: Epidemiology, natural history, diagnosis and treatment; 2007). If not taken care of, PAD may lead to critical limb ischemia, open sores on the feet or legs that become infected by gangrene. The gangrene is then removed surgically, but doctors might have to amputate the extremity all together. Another issue that arises with PAD if it is not under control is the risk of stroke or heart attack. These can cause death to part of the heart or brain, or even death itself. The population more at risk would be smokers, diabetics, people who are obese, those with hypertension or hyperlipidemia, over the age of 50, have a family history of PAD, or those with a high level of homocysteine. If someone does fall into a few of these categories a physician can do a few tests to check for PAD. The doctor will more than likely start off with a physical exam, blood test, and possibly an ultrasound. From there the physician may try an ankle-brachial index, or ABI, which compares the blood pressure of the feet to the blood pressure of the arm. “An American survey of 2174 patients older than 40 years of age used the ankle-brachial index (ABI) as a screening tool, and showed a PAD prevalence of 0.9% between the ages of 40 and 49 years, 2.5% between the ages of 50 and 59 years, ...
A hypothesis that can be made from the patient’s report is that she is suffering from cervical radiculopathy, or a nerve root lesion. Symptoms that describe cervical radiculopathy include: arm pain in a dermatome distribution, pain increased by extension, rotation, and/or side flexion, possible relief of pain from arm positioned overhead, affected sensation, altered hand function, no spasticity, and no change to gait or bowel and bladder function (Magee, 2008, p. 142). These symptoms correlate to what the patient reported as a result of her injury. She stated that her pain is in the posterolateral upper and lower arm with aching and paresthesia in the thumb and index finger, which is in the dermatome pattern of cervical root 5 and 6 (C5, C6) (Magee, 2008, p. 25). She also reports lancinating pain with extension or rotation to the right of her head.
Whatever diabetic stage or type you are at, it is important that you are proactive with your health and treatment. Always be discussing treatments with your doctor or health provider.
Patients whose lesion is in the Cerebrum & Cerebellum will experience loss of balance and coordination, difficulty speaking, and frequent limb trembles. Speech difficulties vary from slurred words, long pause between words, and swallowing problems. Patients whose lesion is in the Motor nerve tracts will experience weakened and stiffened muscles, blurred vision or vision impaired, and urinary problems. Weakened and stiffened muscles causes walking disabilities and painful feeling of muscle spasms in 6 to 10 people. Patients whose lesion is in the Sensory nerve tract will experience sensory alternations, fatigue, cognitive and emotional dysfunction, and loss of sexual interest. Difference sensations experienced are numbness, itching, burning, stabbing, or tearing pains.
Peripheral and central mechanisms involving nerve lesions and their input are substantial when perceiving phantom pain. Due to the impairment of peripheral nerves in the process of amputation, regenerative sprouting of damaged axons occurs and the activity rate of inflamed C-fibres and demyelinated A-fibres spontaneously increases (Flor, 2002). As a consequence of this nerve injury, a neuroma, which is a mass of pruned and tangled axons, may form in the residual limb producing abnormal (ectopic) activity (Katz, 1992). Flor, Nikolajsen and Jenson (2006) proposed that ectopic discharge from a neuroma in the stump illustrates abnormal afferent input to the spinal cord, which is a possible mechanism for unpro...
When a person begins to suffer from Guillain- Barre Syndrome their myelin sheath of their nervous system is being attacked and destroyed by the immune system (NINDS, 2011). The myelin sheath begins to lose its ability to transmit signals rapidly and affectively. Since signals are not getting transmitted to the brain fast enough, a person begins to notice fewer sensory responses from the rest of the body (NINDS, 2011). A person wouldn’t be able to tell right away or at all if an item they are touching is hot, cold, or causing pain. There also wouldn’t be good signal transmission from the brain to the rest of the body (NINDS, 2011). There would be signs of the muscles being unable to respond to the weakened or distraught signals they were receiving. Since the myelin sheath is responsible for transmitting the signals from a long distance, the upper and lower extremities would be the first to show signs of muscle dysfunction.
Although diabetes can be genetic, that doesn’t necessarily mean you’re prone to get it. Controlling and checking your blood sugar helps to treat diabetes, as well as insulin therapy, regular check-ups/monitoring, and exercising. If you’re diabetic, you’ll find that at first, it’s frustrating and time consuming to treat for your diabetes. You’ll get the hang of it soon enough.
Parkinson's Disease has caused problems for many people in this world and plagued the elderly all over the world.Parkinson's disease still puzzles doctors and the causes are unknown. It is known that it is a non-communicable disease and may even be hereditary. Parkinson's disease is thought to be caused by external factors. Most of the cases of this disease are caused by progressive deterioration of the nerve cells, which control muscle movement. Dopamine, one of the substances used in the brain to transmit impulses, is produced in the area of deterioration.
A good test for confirming Guillain-Barre is the nerve conduction test. If there is a sign of demyelination this test will show that the nerve impulse will take a longer time to contract the muscle. Another test for confirming Guillain-Barre would be testing the blood for cortisol. It has shown that patients with Guillain-Barre will have higher levels of cortisol in their plasma (Lescher, 2011). Efferent paralysis is more prevalent than afferent loss; however, in most variations of Guillain-Barre, sensory deficits will occur on both sides, symmetrically. As stated before, it is more severe in hands and feet; this is commonly stated as, glove-and-stocking sensory loss. “This distal pattern of sensory loss is caused by axonal transport, or the dying-back phenomenon, wherein the parts of the axon most affected are those most remote from the cell bodies in the dorsal root ganglia." (Schenkman, M. L.,
Diabetic Neuropathy is a group of neurological disorders caused by nerve damage resulting from the effects of type one and type two Diabetes. There are several types of neurological diseases, each affecting the individual differently. Each disorder presents with it's own symptoms, prognosis, and risks. It is important for the diabetic patient to receive adequate education from their physician on the basic signs and symptoms of neuropathy. Early identification and frequent check ups can impact the effects of neuropathy greatly.