The ear is made up of three parts, the outer ear, the middle ear and the inner ear. The ability to hear is dependent on these three parts of the ear working together, and a problem with any part can cause hearing loss (heaing loss education centre, 2012). The inner ear consists of the cochlea, the auditory hearing nerve and the brain. These are the organs of hearing and balance and convert sound waves into nerve signals. These signals are sent to the brain using a nerve called the vestibulocochlear nerve. Nerve deafness occurs if there is damage to the inner ear and although it is possible to regain some hearing through the help of a hearing device, nerve deafness is often permanent. (deaf websites, 2013)
Damage to the inner ear can be caused by old age, exposure to loud noise, family history or Ménière’s disease.
Ménière’s disease is an incurable disorder with ‘vertigo, tinnitus (ringing, buzzing, noises in the ears) and progressive deafness’ (Nordqvist, 2009) . It affects one in a thousand people and can occur at any age, although it is more common between the ages of forty and sixty. The disease starts in one ear and around twenty five percent of these people will be affected in the other ear at a later date (knott, 2012) .
SYMPTOMS OF MENIERE’S DISEASE
Meniere’s disease usually occurs in attacks of many symptoms and can be unpredictable. The patient may be affected by attacks of these symptoms followed by long periods of time where they are not at all affected. This is referred to as remission. These attacks can last anywhere from twenty minutes to twenty four hours. The main symptoms include vertigo, tinnitus, a feeling of deep pressure inside the ear and hearing loss.
Vertigo is the most common and noticeable sym...
... middle of paper ...
...12 2013].
NHS, 2012. symptoms of menieres disease. [Online]
Available at: http://www.nhs.uk/Conditions/Menieres-disease/Pages/Symptoms.aspx
[Accessed 22 12 2013].
nhs, 2013. Tinnitus. [Online]
Available at: http://www.nhs.uk/Conditions/Tinnitus/Pages/Introduction.aspx
[Accessed 22 12 2013].
NHS, n.d. prochlorperazine maleate. [Online]
Available at: http://www.nhs.uk/medicine-guides/pages/MedicineOverview.aspx?condition=Vertigo and dizziness&medicine=prochlorperazine maleate&preparation=Prochlorperazine 5mg tablets
[Accessed 01 02 2013].
NIDCD, 2013. hearing aids. [Online]
Available at: https://www.nidcd.nih.gov/health/hearing/pages/hearingaid.aspx
[Accessed 02 01 2014].
Nordqvist, C., 2009. what is menieres disease? what causes menieres disease?. [Online]
Available at: http://www.medicalnewstoday.com/articles/163888.php
[Accessed 23 12 2013].
Hearing allows us to take in noises from the surrounding environment and gives us a sense of where things are in relation to us. All those little folds on the outside of the ear, called the tonotopic organization, make it so sound waves in the air are directed to the ear canal, where they can be further processed. Once in the ear, the sound waves vibrate the ear drum, which tell the ear exactly what frequency it is sensing. The vibration of the ear drum is not quite enough to send a signal to the brain, so it needs to be amplified, which is where the three tiny bones in the ear come into play. The malleus or hammer, incus or anvil, and stapes or stirrup amplify this sound and send it to the cochlea. The cochlea conducts the sound signal through a fluid with a higher inertia than air, so this is why the signal from the ear drum needs to be amplified. It is much harder to move the fluid than it is to move the air. The cochlea basically takes these physical vibrations and turns them into electrical impulses that can be sent to the brain. This is...
Flaccid dysarthria results from damage to the lower motor neurons (LMN) or the peripheral nervous system (Hageman, 1997). The characteristics of flaccid dysarthria generally reflect damage to cranial nerves with motor speech functions (e.g., cranial nerves IX, X, XI and XII) (Seikel, King & Drumright, 2010). Lower motor neurons connect the central nervous system to the muscle fibers; from the brainstem to the cranial nerves with motor function, or from the anterior horns of grey matter to the spinal nerves (Murdoch, 1998). If there are lesions to spinal nerves and the cranial nerves with motor speech functions, it is indicative of a lower motor neuron lesion and flaccid dysarthria. Damage to lower motor neurons that supply the speech muscles is also known as bulbar palsy (Pena-Brooks & Hedge, 2007). Potential etiologies of flaccid dysarthria include spinal cord injury, cerebrovascular accidents, tumors or traumatic brain injury (Pena-Brooks & Hedge, 2007). Possible congenital etiologies of flaccid dysarthria include Moebius syndrome and cerebral palsy. Flaccid dysarthria can also arise from infections such as polio, herpes zoster, and secondary infections to AIDS (Pena-Brooks & Hedge, 2007). Additionally, demyelinating diseases such as Guilian-Barre syndrome and myotonic muscular dystrophy can also lead to flaccid dysarthria (Pena-Brookes & Hedge, 2007). The lower motor neuron lesion results in loss of voluntary muscle control, and an inability to maintain muscle tone. Fasciculations, or twitching movements, may occur if the cell body is involved in the lesion (Seikel et. al., 2010). The primary speech characteristics of flaccid dysarthria include imprecise consonant production, hypernasal resonance, breathiness, and harsh voice (...
Mark started losing his hearing when he was about six or seven years old. This was manifested in confusion in music class, misunderstanding the words that the choir was singing, and discombobulation in noisy rooms. Eventually, it was noted that Mark’s hearing was deteriorating.
“Music is perpetual, and only the hearing is intermittent,” wrote the iconic American essayist, poet and philosopher Henry David Thoreau, a lofty proclamation that inspired my focus to help those with hearing loss through restoration. After a winding journey in search for an academic focus, I discovered that audiology is far more than just aiding deaf or hard of hearing individuals, but restoring balance, managing loss through therapy, and discovering new research techniques that may involve auditory neuropathy spectrum disorder. After arriving at my destination, I also learned that it is my responsibility as a future audiologist to be a leader, to work hard toward achieving a better future for myself, and a better world for humanity at large. This vision drives my aspiration to join the University of South Florida’s graduate audiology program this coming fall, and continue my examination of clinical audiology as a member of your community.
Meniere’s disease is an inner ear disorder. People diagnosed with Meniere’s disease experience a variety of symptoms which include a fluctuating hearing loss which can turn permanent, tinnitus or a ringing sound in the ears, ear pressure, and spinning also known as vertigo. Most people with Meniere’s disease only have one ear affected by the disease. Meniere’s disease usually affects people between the ages of 20 and 50, but it can appear at any age. Meniere’s disease is chronic and there is no cure, but there are treatment options people can use to minimize some of the symptoms they might experience (1).
After the sound is processed in the cochlea, the auditory information travels into the brain in order to be interpreted.
Hearing loss is a major global public health issue. Hearnet (2017) defines hearing loss as “a disability that occurs when one or more parts of the ear and/or the parts of the brain that make up the hearing pathway do not function normally” (para. 1). There are many different types of hearing loss, which can have multiple causes, giving each individual experiencing the issue a unique hearing loss case. These types include Auditory Processing Disorders, when the brain has problems processing sound information; Conductive Hearing Loss, a problem with the outer or middle ear which prevents sound making its way to the inner ear; and Sensorineural Hearing Loss, when the Cochlea or auditory nerve is damaged and cannot
The ear houses some of the most sensitive organs in the body. The physics of sound is well understood, while the mechanics of how the inner ear translates sound waves into neurotransmitters that then communicate to the brain is still incomplete. Because the vestibular labyrinth and the auditory structure are formed very early in the development of the fetus and the fluid pressure contained within both of them is mutually dependant, a disorder in one of the two reciprocating structures affects the (2).
The current hypothesis is that one of my genes is a mutated gene, that mutated gene is what is causing my hearing loss. If this is the real reason why I have hearing loss, there is also worry for what other problems does this mutated gene cause. With finding a mutated gene, they will most likely be able to predict how much worse my hearing will get. Another possible but not likely cause is a tumor, currently, I have to get an MRI to make sure that there is no growth inside of my head. If there is a growth, that will lead to some serious issues. The last possible cause is that loud noises have damaged my hearing, but it is even more less likely than a tumor. I am almost never exposed to loud music, concerts, or anything of that nature, which would causes hearing loss. Since I've been losing hearing since I was 5, they have practically ruled that one out because it makes no
symptoms between the ages of 30 and 50, but has been known to show itself in
In our everyday lives, we almost take for granted this idea of balance or equilibrium that is maintained within our bodies. In general, no real thought processes are required. It is only when something is disturbed within our balance system that one is able to take notice of changes in the equilibrium. There may be several different factors that cause a disturbance to our bodies. One major area pertains to dizziness. Dizziness is found to be "the chief complaint in 8 million physician visits a year" (1). Vertigo is one type of dizziness, causing illusions of movement, that is being researched more and more today because of its widespread symptoms.
Often within classroom environments, as well as at home, children learn through visual and auditory perception. Visual and auditory processing are key ways to learn; they are used for recognizing and interpreting information taken from the two senses of sound as well as sight. So clearly it is understood that having this disorder can make it a bit more difficult and troublesome to learn through vision and hearing, but definitely not impossible.
Then, when I was three years old, I had surgery to get a cochlear implant at the University of Minnesota. A cochlear implant is a small device which bypasses the damaged parts of the ear and directly stimulates the auditory nerve. Signals generated by the implant are sent by the auditory nerve to the brain, which recognizes t...
The ear is an organ of the body that is used for hearing and balance. It is connected to the brain by the auditory nerve and is composed of three divisions, the external ear, the middle ear, and the inner ear. The greater part of which is enclosed within the temporal bone.
Hearing is known to be an automatic function of the body. According to the dictionary, hearing is, “the faculty or sense by which sound is perceived; the act of perceiving sound,” (“hearing…”). Hearing is a physical and involuntary act; therefore, unless one is born with a specific form of deafness, everyone has the natural ability to hear sounds. Sounds constantly surround us in our everyday environments, and because we are so accustomed to hearing certain sounds we sometimes don’t acknowledge them at all (or “listen” to them). The dictionary definition of listening is, “to give attention with the ear; attend closely for the purpose of hearing,” (“listening…”). This differs from hearing in that this is a voluntary action, and we have control over what we choose to listen to. As stated by William Seiler and Melissa Beall, “You don’t have to work at hearing; it just happens… Listening, on the other hand, is active and requires energy and desire,” (145).