Familial adenomatous polyposis Essays

  • Familial Adenomatous Polyposis: Case Study

    633 Words  | 2 Pages

    Interesting Facts- v The probability of having polyposis varies from 1 out of 7,000 to 1 out of 22,000 v Polyposis is inherited through reproduction in an autosomal dominant pattern in the chromosomes of the offspring. Works Cited Wehbi, Mohammad, and John M. Carethers. “Familial Adenomatous Polyposis.” : Background, Pathophysiology, Epidemiology. N.p., 6 Mar. 2015. Web. 30 Mar. 2016. Stavro, Steven Atanas. “Result Filters.” National Center for Biotechnology Information. U.S

  • Colon Cancer

    2271 Words  | 5 Pages

    years. Before a true cancer develops, there usually are precancerous changes in the lining of the colon or rectum. These changes might be dysplasia or adenomatous polyps. A polyp is a growth of tissue into the center of the colon or rectum. Some types of polyps (hyperplastic polyps and inflammatory polyps) are not precancerous. However, having adenomatous polyps, also known as adenomas, does increase a person’s risk of developing cancer, especially if there are many polyps or they are large. In contrast

  • Colon Cancer

    659 Words  | 2 Pages

    that cause this cancer, which allows it to be passed down to your family members. The two known inherited colorectal cancer syndromes are hereditary nonpolyposis colorectal cancer (HNPCC) also known as Lynch syndrome. The second one is familial adenomatous polyposis (FAP). Both of these can affect male or female, and the children of people who carry these have a 50% chance of inheriting the disease-causing gene. Lynch syndrome (HNPCC), a change/mutation in certain genes which may allow the formation

  • The Mutation of Cancer Cells

    1753 Words  | 4 Pages

    My sister, Kathy, was diagnosed with cancer in 2013. I was shocked because my sister was always the healthy one among all us girls, the type of cancer, Kathy called colon cancer, Cancer that forms in the tissues of the colon. Most oncogene mutations of indisputable normal genes designate proto-oncogenes. Proto-oncogenes determine the “excellent” genes that usually rule what cell do and the way typically it distribute. Once a factor mutates (changes) into cell, it come back a "hurtful" factor that

  • Gut Microbes Combine To Cause Colon Cancer

    978 Words  | 2 Pages

    How could your gut bacteria cause you colon cancer? According to the article, “Gut Microbes Combine to cause Colon Cancer, Study Suggests” They reported that there is two type of bacteria causing colon cancer according to recent research publications. There are two type of bacteria associated with colon cancer Bacteroides fragilis and E. coli, they are known to produce a layer in the intestine lining. According to this article, E. coli have toxin that can damage DNA of the colon cells. Additionally

  • Genetic Screening for Colorectal Cancer

    3012 Words  | 7 Pages

    cancer.org/cancerinfo/documents/overviews/coloover.asp?ct=10. O’Leary, T. 1999 Molecular diagnosis of hereditary nonpolyposis colorectal cancer. JAMA: Journal of the American Medical Association. 282 (3): 281+. Peterson, G. M. Lessons from familial cancers. Mid-Atlantic Cancer Genetics Network Newsletter. Fall 1999. http://www.macgn.org/nl13e.html. Raab, M. 1998. Genetic shakeup. Working Woman. October 1998. Rogers, J. 1994. Study shows imprinting directly tied to cancer. Johns Hopkins

  • The High Cost of Genetic Engineering

    1197 Words  | 3 Pages

    The High Cost of Genetic Engineering Genetic research on human embryos, in correlation with the human genome, is the key to gene therapy, genetic diagnosis, and even to genetically engineered human beings.  Knowing which gene controls what trait and causes what genetic disease will arm doctors with a powerful tool to treat their patients at the molecular level.  On the other hand, this allows people to possibly manipulate genes to enhance specific traits or create the perfect baby.  Genetic

  • Genetic Testing and Screening

    1882 Words  | 4 Pages

    tests to screen for these genes is becoming possible for a much larger number of diseases. Some of the current DNA tests available diagnose Adult polycystic kidney disease, Alpha-1-Antitrypsin deficiency, familial adenomatous polyposis, hemophilia, Huntington's disease, myotonic dystrophy, and familial breast cancer susceptibility (Munson, 1996). Many more tests are well on the way to being developed. With the possibility o... ... middle of paper ... ...e water. We must carefully look at the benefits

  • The Genetic Screening Debate

    1835 Words  | 4 Pages

    The Genetic Screening Debate Within the past thirty years, researchers have found strong evidence linking genes and disease. The development of predictive genetic tests followed shortly after the isolation of certain candidate genes. Although predictive genetic screening is only available for a handful of diseases, its effects and ramifications have become hotly debated issues in a wide range of areas, from government to religion. The debate began in the 1993 when researchers isolated the BRCA1

  • Identification of Cuminoids with Anticancer Activity

    1447 Words  | 3 Pages

    Numerous epidemiological studies have shown that a diet high in vegetables and fruits is associated with reduced risk of most cancers [1,2]. Since ancient times, herbs and spices have been used for the treatment of various illnesses. They have been used to treat respiratory, gastrointestinal, rheumatic and inflammatory disorders. The beneficial effects of dietary phytochemicals such as curcumin (from turmeric), allicin (from garlic), and resveratrol (from grapes and peanuts) against inflammation

  • Cancer

    2308 Words  | 5 Pages

    Cancer is a genetic disease because it can be traced to alteration within specific genes, but in most cases, it is not an inherited disease. The genetic alterations that lead to most cancers arise in the deoxyribonucleic acid (DNA) of a somatic cell during the lifetime of affected individual. Because of these genetic changes, cancer cells proliferate uncontrollably, producing malignant tumour that invade surrounding healthy tissue. As long as the growth of tumour remains localized, the disease can