Free Atherosclerosis Essays and Papers

Atherosclerosis is a major form of cardiovascular disease which occurs through a processed known as arteriosclerosis in which the arteries become clogged with plaque buildup overtime. Formation of this plaque is due to the damage of endothelial cells (inner cells) that line the walls of arteries. Damage of these cells is triggered by unhealthy practices or other conditions such as smoking, lack of exercise, hypertension, diabetes or high cholesterol levels. As a result harmful material such as fat

Atherosclerosis is a cardiovascular disease characterized by the deposition of materials such as lipids, cholesterol, and proteins such as fibrins in the arteries, as seen in Figure 1 below. This leads to increased resistance to blood flow and causes the stress on the heart to increase. The main risk of atherosclerosis is that it greatly increases the probability of blood clots forming in arteries. Should such clots occur in the carotid or coronary arteries, they can result in strokes or myocardial

Atherosclerosis is defined as the formation of fatty plaques in the walls of arteries, restricting blood flow to deep tissues (Martini). Coronary heart disease came in the public eye in the mid-20th century as a disease of pandemic magnitudes and it continues to be a major cause of death in the western world. The fundamental cause of coronary heart disease is Atherosclerosis. The term “atherosclerosis” is the combination of two Greek words which correctly imply the elements of the lesion. The first

Atherosclerosis is the culprit behind coronary heart disease (CHD) and stroke, which is the most common cause of death worldwide and in the United States10. Among the modifiable risk factors of CHD and stroke is the serum low density lipoprotein level (LDL)8, 11. Several randomized clinical trials have established that reducing the serum LDL level results in a reduction in the future risk of CHD and stroke in a linear relationship, in one study it was estimated that reducing the LDL by 1 % would

Atherosclerosis is a disease in which plaque builds up on the inner walls of the arteries, leading to cardiovascular disease (Gibbons). Arteries are blood vessels that carry oxygen-rich blood to the heart and other locations in the body (Gibbons). The plaque inside of the arteries is made up of fat, cholesterol, calcium, and other substances found in blood (Gibbons). Plaque hardens and narrows arteries over time, limiting the flow of oxygen-rich blood to the organs (Gibbons). Atherosclerosis may

Atherosclerosis, one of the leading causes of death in the country, is a condition in the arteries that is characterized by the deposition of plaque on the arterial walls. While the exact cause of atherosclerosis is unknown, it is a slow, complex disease that begins developing during childhood, and progresses faster with age.1 Certain factors such as smoking, obesity, high cholesterol, high blood pressure, and increased concentrations of sugar in the blood (or diabetes), play a role in the development

Atherosclerosis, the underlying causes of coronary heart disease can affect any artery in the body. Coronary heart disease is essentially the build up of plaque which sticks to the walls of the coronary arteries. The arteries in atherosclerosis is narrowed due to the excessive build up of cholesterol-rich lipids known as plaque around the artery wall (Insull, 2009). Once the plaques around the walls of the arteries become narrow, the flow of blood will be constricted, reducing the amount of blood

Atherosclerosis is a disease that occurs when arteries become blocked, inflamed, or hardened. As a result of this, blood cannot easily pass through the artery, and blood pressure increases. Many people suffer from atherosclerosis as they age, but young people can be affected by atherosclerosis also. There are many preventative steps that can be taken to decrease the risk of atherosclerosis; however, if atherosclerosis does develop in the arteries, medications can be given to help the individual receive

INTRODUCTION Atherosclerosis, a disease that hardens and thickens arterial walls, consists of multiple components such as the following: fat components, cholesterol, and cellular waste products. These components produce a build up in the lining of the arteries.1 Smoking and medical conditions such as elevated triglycerides, elevated cholesterol, and high blood pressure can cause atherosclerosis.1 As a result of the collection of these components, arteries accumulate plaque, which consists of fatty

crisis.1 Increased macrovascular disease, manifesting as premature atherosclerosis, has been well established in other systemic autoimmune rheumatic diseases such as rheumatoid arthritis (RA) and systemic lupus erythematosus (SLE).2,3 A recent systematic review showed that patients with SSc have also an increased carotid intima-media thickness compared to controls.4 Despite this, data on whether these surrogate markers of atherosclerosis translate into an increased risk of MI and stroke events in patients