Ataxic dysarthria is one of several types of dysarthria. “Dysarthria is a group of motor speech disorders resulting from disturbed muscular control of the speech mechanism due to damage of the peripheral or central nervous system” (Hedge, 2001, p. 198). In specific, ataxic dysarthria is a disorder that affects the control of speech production due to damage to the cerebellum or the neural pathways that connect the cerebellum to other parts of the central nervous system. McNeil (1997), states that
1. Medical Conditions/Diagnosis: Pa has a diagnosis of HTN, CAD, hyperlipidemia, seizures, and ataxia. The Pa suffered a stroke 04/26/2013. Pa experience many residual medical complications following the stroke. These conditions cause pain, weakness, fatigue unsteady gait, agitation, ataxia and aphasia is present due to Pa’s stroke. Pa’s spouse supervises Pa when Pa completes daily ADLs because Pa is a fall risk and is not able to stand for more than a few minutes at a time. 2. Skilled medical
I’m filled with pride every time I get to explain that it was my friends who started this program, my friends and I who are making such an impact on the lives of those with Friedreich’s Ataxia. At the root of the problem, FA is a disease most people have never heard about, let alone give any level of importance to. That changes when 1 of the 1 per 50,000 individuals diagnosed is your close friend - it is slowly but surely attacking Raena’s
One of the different stroke is the lateral medulla syndrome. Most of the time it gets misdiagnosed a 50-year-old man who is a non-smoker, non-hypersensitive, non-diabetic showed the history of abrupt commencement of discord of the gait, dysarthria and dysphagia. He is radiologically and clinically diagnosed as a case history of Ischaemic stroke. This subject improved considerably with treatment. Wallenberg is usually caused by obstruction of the cranial articulation of the rear inferior cerebellar
author signaled the usage through elements with his word choices, transitions, and logical connections. The second article summarized analytically is Dark Prophecy: New Test Tells Whom a Crippling Disease Will Hit – and When --- Gene Responsible for Ataxia Has Same Flaw as Those Causing Some Cancers --- the ‘Stutter’ in the DNA by author Marilyn Chase. In this article, I identified the author’s use of the three elements: experiment, correlation, and speculation to support assertions, and analyzed how
poison by his own hand. Socrates drank up, then walked around until he noticed his legs were heavy (Holm, 1997; Scutchfield and Genovese, 1997). Symptoms described by Socrates were: a rapid loss of power of the lower extremities (muscular weakness), ataxia, staggering and trembling. As the effects ascended, there was loss of control of the upper extremities. Total paralysis of the legs and arms followed. There was loss of the power to chew and loss of sensation and the pupils became fixed. If ingested
2.3 Poison hemlock (Conium maculatum) Conium maculatum is a tall, branched, biennial plant, usually 1–2.5m high, and thought to be one of the most toxic members of the family Apiaceae (formerly Umbelliferae) of plant kingdom. The stems are rigid, smooth, and hollow except at the nodes. The plant has a bitter taste and white flower. The leaves are large, triangular; fern like, and alternate on the erect stem. The plant usually grows in waste places where moisture may accumulate and protected from
classical signs and symptoms of Cerebral Palsy are: - 1. Spasticity (Stiff muscles and exaggerated reflexes) (Stern, 2017). 2. Spams (Cerebral Palsy Guide, 2017). 3. Involuntary Movements (facial gestures) (Stern, 2017). 4. Unsteady Gait (Stern, 2017). 5. Ataxia (Lack of muscle co-ordination) (Stern, 2017). 6. Athetosis (Slow writing movement) (Stern, 2017). 7. Rigidity (Stiff muscles with normal reflex) (Stern, 2017). 8. Variation in muscle tone (Either too stiff or too floppy) (Stern, 2017) 9. Decrease
Olivopontocerebellar Atrophy Olivopontocerebellar atrophy (OPCA) was first described in 1900 by Dejerine and Thomas. OPCA is a group of dominant inheritance and sporadic neurological disorders characterized by a chronic, progressive, cerebellar ataxia that begins in middle age. The cerebellum and its connections are the primary sites of the disease in chronic progressive disorders that often occur in familial or hereditary patterns. Postmortem studies indicate an atrophy of the cerebellum, pons
therapy practitioners enable people with SCI to return to productive lives. Rehabilitation interventions for neuromuscular conditions often address motor learning. Therapeutic gains may be mitigated by changes in a person’s condition. Variability in ataxia and disruption of conscious proprioception vibration, and discriminative touch may make it difficult to control for differences. With this impairment, the occupational therapist should identify control parameters that are most important for Tom. These
DRAFTING Proprioception provides an awareness of the body and body positioning without 'continuous reference to consciousness' (Lephart et.al, 1997, p. 131). There are two types of proprioception, being conscious and unconscious. Conscious proprioception concerns joint position sense and kinetic sense (Khasnis & Gokula, 2003). Joint position sense processes joint movement and joint position sensations (Sharp et.al, 1994). These joint sensations provide the awareness of the position of the body and
threatments of the disease. The practical part is an investi... ... middle of paper ... ...of minutes but can reoccure many times a day. The group inculeds paroxysmal paresthesia, trigeminal neuralgia, painful tonic spasms, paroxysmal dysatrias and ataxias, paroxysmal diplopia, paroxusmal dyskinesia, facial myokymia. Here we can also include the symptom of Lhermitte. 11. Tiredness Another of the common sympotms. Generally is described as tirdness not related to physical exaustion. There is no connection
Introduction and Biology Kuru is a progressive, fatal, brain disease that was discovered in the 1950s. This disease was discovered by the American Physician Carleton Gajdusek among the people of eastern highlands of New Guinea. Kuru is a disease caused by the consumption of contaminated brain tissue. Kuru is one of a group of rare brain diseases called the transmissible spongiform encephalopathies (TSE’s). TSE’s, leads to forming tiny holes in the brain tissue giving it a spongy look. History It
Virus Classification: Order: Mononegavirales Family: Parvoviridae Subfamily: Parvovirinae Genus: Parvovirus Species: Feline panleukopenia virus Also called feline infectious eneritis, feline “distemper”, and feline ataxia or incoordination. Examples of other viruses belonging to the same genus as Feline panleukopenia virus include Canine parvovirus type 2, Porcine parvovirus, Mink enteritis virus, and Raccoon enteritis virus. Virus Structure: Feline panleukopenia
This paper will discuss what Guillain-Barre Syndrome is. It will also discuss what causes Guillain-Barre Syndrome and how to treat it. This paper will go into detail about the pathophysiology of GBS. The information in this paper was gathered from the Williams/Hopper book used for the nursing program and an article titled “Recent developments and future directions in Guillain-Barre syndrome” written by Helmar C Lehmann Richard A.C. Hughes, Bernd C Kieseier, and Hans-Peter Hartung. Also used for
Encephaloceles Encephaloceles are rare disorders that are caused by a neural tube defect in which the bones of the skull fail to close completely. This failure to close allows cerebral spinal fluid, brain tissue, and the meninges to push through the gap and create a sac-like formation protruding from the skull. Encephaloceles differ from meningoceles in that meningoceles contain only the meninges and cerebral spinal fluid, whereas encephaloceles involve brain tissue being pushed through the sac
Prion Diseases Abstract Transmissible spongiform encephalopathies (TSEs) are neurodegenerative diseases that are thought to be caused by the misfolding of prion proteins. Prions are able to replicate in the absence of nucleic acids. TSEs include: scrapie, bovine spongiform encephalopathy, Creutzfeldt-Jakob disease, kuru, Gerstmann-Straussler-Scheinker disease, and Fatal Familial Insomnia. They can affect many different animals, including humans. Currently, there are no ways to diagnose, treat
Physical Therapy, 92.6 p.799 Retrieved from http://dx.doi.org.ezp-02.lirn.net/10.2522/pt;.20110056 " Multiple Sclerosis (MS) is associated with a wide range of impairments in the body's functions, including motor deficits such as muscle tone, ataxia and abnormal balance, and sensory deficits"(p.799). Boissonault, W. G.; & Lovely, K. (2016, November). Hospital- based out patient direct access to physical therapist services: current status in Wisconsin. Physical Therapy, 96(p.11), 1695+. Retrieved
Low-Density Lipoprotein Receptor VLDLR Cause Cerebellar Hypoplasia and Quadrupedal Locomotion in Humans.” Proceedings of the National Academy of Sciences 105.11 (2008): 4232–4236. Türkmen, Seval et al. “CA8 Mutations Cause a Novel Syndrome Characterized by Ataxia and Mild Mental Retardation with Predisposition to Quadrupedal Gait.” Ed. Gregory S. Barsh. PLoS Genetics 5.5 (2009): e1000487.
The Meningeal Worm Infestation of up to 20 meningeal worms has been discovered in a single deer’s subdural cavity. The white tail deer is the preferred host, but they rarely ever suffer from any sickly or neurological problems from this type of worm. We will see severe signs in llamas and alpacas; these are the two animals that can become infected with it frequently. The meningeal worm can cause damage to the central nervous system and could result in death so it is important to try and catch