Heart Failure is classified as a complex disease resulting from any functional or structural cardiac disorder impairing the competence of the ventricle to fill or expel blood (Porth, 2010). Heart failure is a condition where the heart is unable to pump enough blood to meet the demand of the body’s need. In contrast to a cardiac arrest, the heart does not stop to beat; it becomes weaker, usually over several months or years occurrence. The heart’s inability to pump adequate blood to fill the chambers. Subsequently, this provokes fluids to accumulate in the lungs and tissues, causing profusion. Attempts are made by the body to recompense for the heart failing either on the left or right side. In whichever forms of heart failure, the heart is …show more content…
This failure in the heart’s ability as a pump may be due to multiple reasons and is classed into two main categories: systolic and diastolic heart failure. Systole and diastole are two different events of heart contraction. Systole is part of the cardiac cycle when the ventricles are contracting; blood is driven through the semilunar valves out into systemic circulation, from the left ventricle, or into pulmonary circulation, from the right ventricle (Bailey, 2012). Also during this event, the atria are in a period of “relaxation”, or filling. The AV (atrioventricular) valves are closed during atrial relaxation. Diastole occurs when ventricles are in a period of filling. The semilunar valves are closed to block blood from moving into circulation. The atria are contracting, moving the blood through the AV valves into the ventricles. The origin of heart failure occurs when the heart is not able to pump the whole blood volume out into circulation or if it is unable to fill the ventricle entirely (Lehne, …show more content…
The enlargement in ventricles is owed to an elevated end-systolic volume. If the heart is not able to sufficiently pump the expected volume of blood with each contraction, which in a normal healthy heart is 50-60%, there will be a residual volume left in the heart after every pump (Heart Healthy Women, 2012). Upon the next period of filling, the heart will receive the exact amount of blood volume from the atria joined with that residual volume from the earlier contraction. This will cause the ventricles to dilate to meet the wants of this increase in volume. Thus this causes the walls of the ventricles to stretch and become thin and weak. Also the muscle layer of the heart, the myocardium, will stretch and not be able to adequately make a absolute and forceful contraction to push blood from the ventricles (Lehne, 2010). Systolic heart failure is more than twice as common in men then in women (CDC, 2012). Common causes of systolic heart failure mostly include coronary artery disease, mainly in women, and hypertension, predominantly in males. Diastolic heart failure (also defined as right side heart failure) is far more popular in females and is characterized by an incompetence of the ventricles to adequately fill with enough blood volume. This diminish in stroke
4. Right Ventricular hypertrophy (RVH) – In a normal heart, the left ventricle has a rather thicker wall than the right due to the fact that it has to pump oxygenated blood to the body as opposed to the right ventricle which only needs to pump deoxygenated blood to the lungs. However, Tetralogy of Fallot causes an enlargement of the right ventricular muscle due to the pulmonary stenosis in the pulmonary
When a muscle contracts and relaxes without receiving signals from nerves it is known as myogenic. In the human body, the cardiac muscle is myogenic as this configuration of contractions controls the heartbeat. Within the wall of the right atrium is the sino-atrial node (SAN), which is where the process of the heartbeat begins. It directs consistent waves of electrical activity to the atrial walls, instigating the right and the left atria to contract at the same time. During this stage, the non conducting collagen tissue within the heart prevents the waves of electrical activity from being passed directly from the atria to the ventricles because if this were to happen, it would cause a backflow. Due to this barrier, The waves of electrical energy are directed from the SAN to the atrioventricular node (AVN) which is responsible for transferring the energy to the purkyne fibres in the right and left ventricle walls. Following this, there is a pause before the wave is passed on in order to assure the atria has emptied. After this delay, the walls of the right and left ventricles contract
The normal Mitral Valve controls blood flow between the upper (left atrium) and lower chamber (left ventricle) of the left side of the heart. The mitral valve allows blood to flow from the left atrium into the left ventricle, but not flow the other way. With each heartbeat, the atria contract and push blood into the ventricles. The flaps of the mitral and tricuspid valves open to let blood through. Then, the ventricles contract to pump the blood out of the heart. The flaps of the mitral and tricuspid valves close and form a tight seal that prevents blood from flowing back into the atria (nhlbi.nih.gov).
It occurs because of repetitive electrical activity. This can occur in a patient with early or late heart failure, because there is damage to the heart tissue and the heart beats faster to try to supply the body with blood. Recommended treatment is elective cardioversion. Drugs used include an antidysrhythmic such as Mexitil or Sotalol (Ignatavicius &Workman, p. 728-729).
O’Rourke [13] describes the pulse wave shape as: “A sharp upstroke, straight rise to the first systolic peak, and near-exponential pressure decay in the late diastole.” Arteries are compliant structures, which buffer the pressure change resulting from the pumping action of the heart. The arteries function by expanding and absorbing energy during systole (contraction of the cardiac muscle) and release this energy by recoiling during diastole (relaxation of the cardiac muscle). This function produces a smooth pulse wave comprising a sharp rise and gradual decay of the wave as seen in Figure 5. As the arteries age, they become less compliant and do not buffer the pressure change to the full extent. This results in an increase in systolic pressure and a decrease in diastolic pressure.
The thickening of the muscle cells do not necessarily have to change the size of the ventricles, but can narrow the blood vessels inside the heart. Hypertrophic cardiomyopathy can be grouped into two categories: obstructive HCM and non-obstructive HCM. With obstructive HCM, the septum (the wall that divides the left and right sides of the heart) becomes thickened and blocks the blood flow out of the left ventricle. Overall, HCM usually starts in the left ventricle. HCM can also cause blood to leak backward through the mitral valve causing even more problems. The walls of the ventricles can also become stiff since it cannot hold a normal amount of blood. This stiffening causes the ventricle to not relax and entirely fill with
The blood circulates through coronary arteries and then to smaller vessels into cardiac muscle (myocardium). The blood flow is influenced by aortic pressure, which increases in systole, and the pumping activity of the ventricles. When the ventricle contracts, in systole, the coronary vessels are compressed by the contracted myocardium and partly blocked by the open aortic valve therefore the blood flow through the myocardium stops.
This systematic review conducted by Takeda A, Taylor SJC, Taylor RS, Khan F, Krum H, Underwood M, (2012) sourced twenty-five trials, and the overall number of people of the collective trials included was 5,942. Interventions were classified and assessed using the following headings.-
Systolic and Diastolic are the two types of heart failure. Systolic dysfunction occurs when the heart muscle doesn't contract with enough force, which means there is less oxygen-rich blood that is pumped throughout the body. Diastolic dysfunction is when the heart contracts normally, but the ventricle does not relax properly, reducing the amount of blood that can enter the heart and raising the blood pressure in the lungs. Heart failure is a progressive condition and can worsen over time. There are four stages of heart failure that have been classified by the AHA and ACC.
Hypertrophic cardiomyopathy is an inherited disease that affects the cardiac muscle of the heart, causing the walls of the heart to thicken and become stiff. [1] On a cellular level, the sarcomere increase in size. As a result, the cardiac muscles become abnormally thick, making it difficult for the cells to contract and the heart to pump. A genetic mutation causes the myocytes to form chaotic intersecting bundles. A pathognomonic abnormality called myocardial fiber disarray. [2,12] How the hypertrophy is distributed throughout the heart is varied. Though, in most cases, the left ventricle is always affected. [3] The heart muscle can thicken in four different patterns. The most common being asymmetrical septal hypertrophy without obstruction. Here the intraventricular septum becomes thick, but the mitral valve is not affected. Asymmetrical septal hypertrophy with obstruction causes the mitral valve to touch the septal wall during contraction. (Left ventricle outflow tract obstruction.) The obstruction of the mitral valve allows for blood to slowly flow from the left ventricle back into the left atrium (Mitral regurgitation). Symmetrical hypertrophy is the thickening of the entire left ven...
Once the atrium contracts, blood cannot flow back into or enter the atria because the openings of the great veins have been narrowed by pressure. The ventricles are now filled with blood, accomplishing end-diastolic volume, which is another term for how much blood your ventricles can contain while your body is at rest. The next phase is early ventricular systole. Now that all the blood is in your ventricles, it must continue onward to the arterial trunk.
The heart serves as a powerful function in the human body through two main jobs. It pumps oxygen-rich blood throughout the body and “blood vessels called coronary arteries that carry oxygenated blood straight into the heart muscle” (Katzenstein and Pinã, 2). There are four chambers and valves inside the heart that “help regulate the flow of blood as it travels through the heart’s chambers and out to the lungs and body” (Katzenstein Pinã, 2). Within the heart there is the upper chamber known as the atrium (atria) and the lower chamber known as the ventricles. “The atrium receive blood from the lu...
Just as breast cancer is killing our African American women, heart disease is also one of the major diseases killing our women. Heart disease is one of the nation’s leading causes of death in both woman and men. About 600,000 people die of heart disease in the United States (Americas heart disease burden, 2013). Some facts about heart disease are every year about 935,000 Americans have a heart attack. Of these, 610,000 are a first heart attack victim. 325,000 happen in people who have already had a heart attack. Also coronary heart disease alone costs the United States $108.9 billion each year. This total includes the cost of health care services, medications, and loss of productivity. Deaths of heart disease in the United States back in 2008 killed about 24.5% of African Americans.
Most often the disease starts in the left ventricle, and then often spreads to both the atrium and right ventricle as well. Usually there will also be mitral and tricuspid regurgitation, due to the dilation of the annuli. This regurgitation will continue to make problems worse by adding excessive volume and pressure to the atria, which is what then causes them to dilate. Once the atria become dilated it often leads to atrial fibrillation. As the volume load increases the ventricles become more dilated and over time the myocytes become weakened and cannot contract as they should. As you might have guessed with the progressive myocyte degeneration, there is a reduction in cardiac output which then may present as signs of heart failure (Lily).
The heart is a pump with four chambers made of their own special muscle called cardiac muscle. Its interwoven muscle fibers enable the heart to contract or squeeze together automatically (Colombo 7). It’s about the same size of a fist and weighs some where around two hundred fifty to three hundred fifty grams (Marieb 432). The size of the heart depends on a person’s height and size. The heart wall is enclosed in three layers: superficial epicardium, middle epicardium, and deep epicardium. It is then enclosed in a double-walled sac called the Pericardium. The terms Systole and Diastole refer respectively and literally to the contraction and relaxation periods of heart activity (Marieb 432). While the doctor is taking a patient’s blood pressure, he listens for the contractions and relaxations of the heart. He also listens for them to make sure that they are going in a single rhythm, to make sure that there are no arrhythmias or complications. The heart muscle does not depend on the nervous system. If the nervous s...