The Effects Of Exercise On Homeostatic Respiration

Homeostasis is the maintenance of a steady state in the body despite external changes. Multiple activities and occurrences can offset the homeostatic equilibrium. In this instance, the effects of exercise on homeostasis were tested. Exercise is a stressor on the body that initializes an automatic negative feedback response system in order to return the body to its homeostatic state by regulating the variable parameters. For the experiment, the subjects were instructed to perform an exercise of their choice consistently and at a manageable pace for six minutes. Their blood pressure, oxygen saturation, heart rate, skin color and the level of perspiration were recorded before exercising and every two minutes for six minutes, and then a minute

This is a great depiction of the body attempting maintain its homeostatic state. Cellular respiration is constantly occurring in the body so that cells have enough energy to perform competently. Since exercise utilizes muscle groups as much as 15 times more than at rest, cellular respiration during exercise happens at a more rapid pace than usual. The byproduct of cellular respiration is carbon dioxide, water, a form of energy (ATP) and thermal energy. Thermal energy leads to the increase of internal body temperature. As nerves (the receptors) send oxygen rich blood flows to the brain, the hypothalamus (control center) releases a signal that sends the blood closer to the skin, where there are pores ( the effectors) so that some of that heat energy can be released though

The averaged trend line for blood pressure, shows a gradual increase from 106.6 systolic at rest to the 137.6 systolic peak at four minutes. From there it begins decreasing in a fashion that mirrors the incline. At two minutes the BP was 126.4 and at 6 minutes it is 125.6 after hitting the highest BP at four minutes. This is a somewhat unexpected result as the blood pressure should have continued rising until it capped off where an adequate amount of blood was dissipating throughout the body. Assuming that the cap off point for this data set was at four minutes (137.6), the heart rate should have remained relatively constant until the subject stopped exercising, leading to a slower blood pressure. It is plausible to state that exhaustion is a factor, therefore the steady pace dropped, and the blood pressure