Cancer Essay

explanatory Essay
1669 words
1669 words

Cancer has been seen in humans as one the most potentially fatal disease for thousands of years and only in the recent couple of hundred years have we discovered that most information necessary to bring us to today’s understanding and knowledge (Kenny 2007, Weinberg 1996) was achieved by extensive research of cells, DNA, and epidemiology studies. As we know, currently cancer is acknowledged as having over a hundred different diseases, and is known to be the result of mutations of the genes and almost similar DNA which are responsible for the amount of cell division and production (Kenny 2007). Restraint of cell growth modulators can be a direct lead and result of certain tumours being developed and subsequently allow these tumours to acquire the ability to attack and occupy the bloodstream and essentially be able to travel via the bloodstream to other parts and organs in human bodies which is known as metastasis (Loeb et Al 2003). Once this has occurred , the cancer is then categorized as malicious and becomes a dangerous and serious threat to the carrier (Weinberg 1996). In this essay I will describe and explain the process of this and how our genes mutate and lead to metastasis of cancer cells. Processes of exogenous and endogenous are likely to result in mutated genes leading towards cancer. (Schulz 2005, Knowles & Selby 2005). There are three known classifications of carcinogens, which are described to be cancer-causing agents based on the fact that they directly boost effects and thus cause mutations of the alterations already in place. The three classifications include: Chemical carcinogens which can be related to cigarettes and the mutations created as a result of the chemicals involved; Physical carcinogens which involves... ... middle of paper ... ...of not only them but also other things. (Schulz 2005). Finally, in conclusion, gene mutations and alterations affect the body’s ability to control the rate of cell division, which therefore defines cancer the direct result of gene mutation. Cells affected by mutations are exposed to the reproduction into a tumour. Once the tumour expands in growth, more and more mutations occur in subsequent cell divisions and eventually become a dysplasia and instigates invasion into surrounding tissues. In simpler terms, the cancer is spread to other organs and parts of the body. Following this, metastasis occurs resulting in the cancer spreading through the person and/or animals blood stream and/or lymphatic systems and forms other colonies which thus secretes other harmful organelles, and also disrupts normal bodily function, and possibly leading to and eventuating in death.

In this essay, the author

  • Explains that cancer has been seen in humans as one of the most potentially fatal diseases for thousands of years and is the result of mutations of genes and almost similar dna responsible for cell division and production.
  • Explains that there are three known classifications of carcinogens, which are cancer-causing agents based on the fact that they directly boost effects and thus cause mutations.
  • Explains that dna contains all the information we need for our bodies to function and thus develop. genes, which contain dna, act as instructions in order to create protein molecules.
  • Explains that microrna genes are responsible for the regulation of gene expression. research and studies have shown that when certain changes occur in these genes, they are closely related to oncogenes and tumour suppression cells.
  • Explains that proto-oncogenes encrypt proteins responsible for cell division control and production and cell death, or apoptosis.
  • Explains that proto-oncogenes are intended to encourage cell division when necessary. tumour suppression genes are merely the opposing force.
  • Explains that hyperplasia occurs when cells are excposed to a mutation of genes that are generally liable for the cell production.
  • Explains that metastasis initiates growth on or near organs, which as a result interferes with its function. gene mutations and alterations affect the body's ability to control the rate of cell division.
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