First, a person with amnesia may work with an occupational therapist to learn new information to replace what was lost, or to use intact memories as a basis for taking in new information. Memory training may also include a variety of strategies for organizing information so that it 's easier to remember and for improving understanding of extended conversation. With some training and practice, even people with severe amnesia can use these electronic organizers to help with day-to-day tasks. For example, smartphones can be programmed to remind them about important events or to take medications. Low-tech memory aids include notebooks, wall calendars, pill minders, and photographs of people and places.
Basically, this perspective is almost the same as biological, but cognitive is based more on memory and how humans are able to process, store and retrieve information and then use the information to solve problems. In Saul McLeod’s articlem” Cognitive Psychology,” the author states,” Even the same memories from different people will be processed differently. Two individuals who went through the same events will also have different memories of those events. As a result, every individual will have a different method of action through the cognitive perspective”(McLeod, 1). What this means is that amnesia can wipe any memory away no matter how long the memory has been stored inside the brain.
Organic causes may include brain damage through injury, or the use of specific drugs - usually sedative drugs. Amnesia may be one of the symptoms of some degenerative brain diseases, such as Alzheimer's disease. Functional causes are psychological factors, such as defense mechanisms. People with amnesia also find it hard to imagine the future, because our constructions of future scenarios are closely linked to our recollections of experiences. Being a little forgetful is completely different to having amnesia.
In old age you may forget a few things here and there, but it is only when the symptoms affect the person’s daily life that it can be called Dementia. The exact cause of Dementia is damage to the brain cells; it affects the way the brain cells are able to communicate with one another. When the cells aren’t able to communicate, it commonly disturbs thinking, behavior, and feelings. Synapse or neuron to neuron functioning is dismal which results in confusion and communication problems. Many things can cause damage to the brain cells such as, diseases that cause deterioration in the brain like Alzheimer’s and Parkinson’s.
The Predicament with Amnesia Amnesia is a type of memory loss that one develops in certain situations. It does not mean, however, that one forgets their self-identity. It states that those who have amnesia only have the trouble of acquiring new memories or forming new memories. Some causes of amnesia are damage to the brain structures that form the limbic system, which is where your emotions and memories are controlled, those who have Alzheimer’s disease, seizures, strokes, tumors in the area of the brain that controls memory, and also alcohol abuse. There are two main different types of amnesia.
The person may be able to recall events in the distant past but not yesterday or today. If the amnesia is caused by alcohol abuse, it is a progressive disorder, and there are usually neurological problems like uncoordinated movements and loss of feeling in the fingers and toes. Once these problems occur, it may be too late to stop drinking. In contrast, psychologically based amnesia is almost always temporary. This type of memory loss may be triggered by a traumatic event with which the mind can not deal.
Multiple-System Memory Theory (Squire, 2004) is the idea that long term memory is composed of individual systems that are independent of one another and handle different types of memories, i.e. declarative, non-declarative. Studying people with deficits, as in amnesia, allows us to observe the effect of brain damage on certain areas of memory, which would be impossible on people of healthy disposition. Amnesia is “a fundamental deficit in relational (declarative) memory processing” (Althoff, Cohen, Ryan & Whitlow, 2000) which is thought to be caused by brain damage to the limbic system including the medial temporal lobe. Amnesic patients tend to be impaired in declarative memory (explicit) but can show intact functioning in non-declarative memory (implicit) (Fleischman, Gabrieli, Keane, Morrell & Reminger, 1995).
The behavioral treatments will allow the patients to undergo a treatment that will give support for the patient and the family (Fisher & Carstensen, 1990; Mintzer et al., 1997; Teri et al., 1997). Treatments of drugs are not supported by clinician due to bad symptoms (Sultzer et al., 2008). It is said that most patients that have dementia that are treated with such anti-psychotics occur greater issues of aggression and mental health. Deaths are more triggered when taken meds. Patients with dementia usually loss all functioning skills once that is lost there is no medications or treatments that will be able to help them gain control or gain back
Memory can fail in numerous ways, from the common examples above to more devasting problems, like Alzheimer's disease. B. Fortunately, brain problems like Alzheimer's can be delayed or even prevented. C. In order to understand how human memory fails, we need a basic understanding of how it works. Body I. Memory is a complex process that takes place primarily in the brain.
The frontal lobe of the brain is responsible for, simply put, impulse control, judgment, memory, language, and much more. When a MS patient has a brain lesion, or a location of the brain that has been heavily damaged, specific functions are affected. The above researchers developed a study to look at the extents of the effects of MS lesions, specifically the white matter of the frontal lobe. In past research, it has been suggested that there may be a threshold where dysfunction of cognitive processes begins. They past research was also deficient in its attempts at looking at frontal lobe lesions, however laid the groundwork that allows for thinking lesion size and location do in fact make a difference in the cognitive differences of MS patients.