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Abstract summary of hypertension
Abstract summary of hypertension
Abstract summary of hypertension
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AIRWAY:
Mrs. Kelly’s s airway is patent. No signs of airway obstruction demonstrated by talking in full sentences. However, Maintaining airway by keeping the patient on an upright position will help eliminate the risk for potential risk of airway obstruction (Dao Le, L.K, 2016). It is a low priority nursing intervention in this case study.
BREATHING:
Mrs. Kelly is Tachypneic as evidence of shortness of breathes due to lack of blood oxygen in the blood called hypoxemia. In which the body is trying to compensate by breathing more. Hypoxemia is taken from an artery to detect the oxygen level in the blood (arterial blood gas). Her ABG results showed a 75 mmHg (normal 80-100mHg). Hypoxemia has many reasons, most often related to respiratory disorders,
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Because the heart is failing to pump enough blood to support the blood supply of the vital organs there will be an increase of systemic vascular resistance, thereby decreasing supply of blood going to non-vital organs such skin but increasing blood flow to vital organs. Her bilateral oedema represents fluid leakage from the blood and lymphatic vessels into the soft tissues. The feet and ankles predisposed to oedema because of gravity, its stimulate pooling of fluids in the areas of the body. This indicates Mrs. Kelly is having a systemic disorder such as heart failure (Evers, Vaneker, & Biert, …show more content…
CHEST XRAY
Chest x-ray is useful in determining the presence of cardiomegaly, pulmonary oedema, pleural effusions, and other criteria of heart failure
It is often used in patients having chest pain to know if there is a presence of pulmonary congestion or cardiac enlargement. It will help to evaluate the finding and prognosticate of early and late mortality of myocardial infarction.
In this case study Mrs Kelly has pulmonary vascular congestion and pulmonary oedema. The fluids in the air spaces and the parenchyma of the lungs accumulate, may cause to impaired gas exchange and lead to respiratory failure, affecting left ventricle of the heart to adequately remove blood from the pulmonary circulation, compromising the cardiac output. This pulmonary oedema can lead to respiratory failure, cardiac arrest due to hypoxia that can lead to death (Morales, Prediletto, Rossi, Catapano, Lombardi, & Rovai, 2012).
The size of the heart is still normal because the cardiomediastinal contours are usually normal in the onset of myocardial infarction.
2. ARTERIAL BLOOD GAS (ABG)
It measures the acidity and the levels of oxygen and carbon dioxide in the blood from an artery. This analysis is used to detect how the oxygen from lungs move in the blood and how carbon dioxide removed from the
The risk factors that Jessica presented with are a history that is positive for smoking, bronchitis and living in a large urban area with decreased air quality. The symptoms that suggest a pulmonary disorder include a productive cough with discolored sputum, elevated respiratory rate, use of the accessory respiratory muscles during quite breathing, exertional dyspnea, tachycardia and pedal edema. The discolored sputum is indicative of a respiratory infection. The changes in respiratory rate, use of respiratory muscles and exertional dyspnea indicate a pulmonary disorder since there is an increased amount of work required for normal breathing. Tachycardia may arise due to the lack of oxygenated blood available to the tissue stimulating an increase in heart rate. The pedal edema most probably results from decreased systemic blood flow.
Both tests are very useful for assessing the pulmonary and cardiac system health of the individual being examined. This involves examining the individual’s response to the test by assessing their BP, HR, oxygen intake abilities, and using this information to infer whether they have some type of internal impairment. This includes
Additionally, some of the general diagnostic and pulmonary function tests are distinct in emphysema in comparison to chronic bronchitis. In the case of R.S. the arterial blood gas (ABG) values are the following: pH=7.32, PaCO2= 60mm Hg, PaO2= 50 mm Hg, HCO3- = 80mEq/L. R.S.’s laboratory findings are indicative of chronic bronchitis, where the pH and PaO2 are decreased, whereas PaCO2 and HCO3- are increased, when compare to normal indices. Based on the arterial blood gas evaluation, the physician can deduce that the increased carbon dioxide is due to the airway obstruction displayed by the hypoventilation. Furthermore the excessive mucus production in chronic bronchitis hinders proper oxygenation leading to the hypoxia. On the other hand, in emphysema the arterial blood gas values would include a low to normal PaCO2 and only a slight decrease in PaO2 which tend to occur in the later disease stages.
Chronic obstructive pulmonary disease or COPD is a group of progressive lung diseases that block airflow and make it hard to breathe. Emphysema and chronic bronchitis are the most common types of COPD (Ignatavicius & Workman, 2016, p 557). Primary symptoms include coughing, mucus, chest pain, shortness of breath, and wheezing (Ignatavicius & Workman, 2016, p.557). COPD develops slowly and worsens over time if not treated during early stages. The disease has no cure, but medication and disease management can slow its progress and make one feel better (NIH, 2013)
HENDERSON, Y (1998) A practical approach to breathing control in primary care. Nursing Standard (JULY) 22 (44) p41
An electrocardiogram (ECG) is one of the primary assessments concluded on patients who are believed to be suffering from cardiac complications. It involves a series of leads attached to the patient which measure the electrical activity of the heart and can be used to detect abnormalities in the heart function. The ECG is virtually always permanently abnormal after an acute myocardial infarction (Julian, Cowan & Mclenachan, 2005). Julies ECG showed an ST segment elevation which is the earliest indication that a myocardial infarction had in fact taken place. The Resuscitation Council (2006) recommends that clinical staff use a systematic approach when assessing and treating an acutely ill patient. Therefore the ABCDE framework would be used to assess Julie. This stands for airways, breathing, circulation, disability and elimination. On admission to A&E staff introduced themselves to Julie and asked her a series of questions about what had happened to which she responded. As she was able to communicate effectively this indicates that her airways are patent. Julie looked extremely pale and short of breath and frequently complained about a feeling of heaviness which radiated from her chest to her left arm. The nurses sat Julie in an upright in order to assess her breathing. The rate of respiration will vary with age and gender. For a healthy adult, respiratory rate of 12-18 breaths per minute is considered to be normal (Blows, 2001). High rates, and especially increasing rates, are markers of illness and a warning that the patient may suddenly deteriorate. Julie’s respiratory rates were recorded to be 21 breaths per minute and regular which can be described as tachypnoea. Julies chest wall appeared to expand equally and symmetrical on each side with each breath taken. Julies SP02 levels which are an estimation of oxygen
On admission, a complete physical assessment was performed along with a blood and metabolic panel. The assessment revealed many positive and negative findings. J.P. was positive for dyspnea and a productive cough. She also was positive for dysuria and hematuria, but negative for flank pain. After close examination of her integumentary and musculoskeletal system, the examiner discovered a shiny firm shin on the right lower extremity with +2 edema complemented by severe pain. A set of baseline vitals were also performed revealing a blood pressure of 124/80, pulse of 87 beats per minute, oxygen saturation of 99%, temperature of 97.3 degrees Fahrenheit, and respiration of 12 breaths per minute. The blood and metabolic panel exposed several abnormal labs. A red blood cell count of 3.99, white blood cell count of 22.5, hemoglobin of 10.9, hematocrit of 33.7%, sodium level of 13, potassium level of 3.1, carbon dioxide level of 10, creatinine level of 3.24, glucose level of 200, and a BUN level of 33 were the abnormal labs.
The two assessment tools chosen are the 12 lead ECG and blood tests. The 12 lead ECG provides more detailed views and angles of the hearts electrical activity in both horizontal and frontal planes. The 12 lead has 6 limb leads and the other 6 for chest leads (Phalen & Aehlert, 2006).
The cardiovascular system keeps blood circulating throughout our bodies constantly, and without it, many of the activities that we do would not be possible! The heart is the main organ of the cardiovascular system because it pumps the blood, and also a major organ of the human body because of how it is able to allow our bodies to work. Unfortunately, the heart may fail to function at times when blood flow is reduced as a result of blockage. This is more commonly known as a heart attack, or myocardial infarction. According to “Heart Disease Statistics,” approximately 715, 000 Americans suffer a heart attack every year . This deadly disorder of the heart can be diagnosed from symptoms, as well as from imaging and tests, and it is possible to
Diagnosis include a pulmonary function test, a test which helps measure the lungs ability to exchange oxygen and carbon dioxide. This type of test is performed with a special machine called spirometry (Mayo Clinic, 2011). A spirometry determines how well the lungs intake, hold, and utilize the air and can even detect the severity of lung disease and determine whether the disease has decreased airflow or a disruption of airflow has occurred. Another device used is a peak flow monitor (PFM). A device that measures the speed at which an individual can blow air out of lungs (Mayo Clinic, 2011). A doctor can diagnose a patient with symptoms that correspond to emphysema, such as a cough that doesn’t go away, coughing up a large sum of mucus, shor...
The physician’s notes indicated a temperature of 98 degrees F, an elevated heart rate and respiratory rate, and low blood pressure. When the amount of oxygen available for the heart is low, it puts pressure on the heart and causes the heart rates to increase. To compensate for the low amount of oxygen the respiratory rate also increases to enable the intake of more oxygen that is be available for the body.
In the case study it is the left lung that is in distress, and as the pressure increases within the left lung it can cause an impaired venus return to the right atrium (Daley, 2014). The increased pressure can eventually affect the right lung as the pressure builds in the left side and causes mediastinal shift which increases pressure on the right lung, which decreases the patients ability to breath, and diffuse the bodies tissues appropriately. The increase in pressure on the left side where the original traum... ... middle of paper ... ... 14, January 29).
The patient has high temperature-sign of fever, a very fast pulse rate (tachycardia), and chest wheezing when listened to using a stethoscope (Harries, Maher, & Graham, 2004, p.
While taking care of my patient on the cardiothoracic intensive care unit, I assisted another nurse who was helping her seventy-one year old patient ambulate to promote circulation and decrease the patient’s chance of developing pneumonia. It also helps the patient to build strength and confidence after such a major surgery like this patient underwent. This patient had come in with non-ischemic cardiomyopathy and had a history of cocaine and alcohol abuse, atrial fibrillation, mitral regurgitation, and hypertension. She had a left ventricular assistive device placed, and ten days after the device had been placed, she was diagnosed with H1N1, had a tracheostomy performed, and was placed on the ventilator. Since she had to wear a mask when outside of her room and had a tracheostomy, it was really difficult to understand the patient’s needs, and this was very concerning to me.
Patient will display adequate gas exchange as evidence by SaO2 values and respiratory rate consistent with baseline.