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...omatic L265P mutation that is found in other kinds of blood cancers. Patients with this disorder have increased levels of IgM in the blood and include diffuse large B cell lymphoma and marginal zone lymphoma. Mutations in NEMO, a protein involved in the MyD88 pathway, causes a disease called X-linked hypohidrotic ectodermal dysplasia and immunodefiency, or also known as NEMO deficiency which can be characterized by development defects and immunodeficiency. In a situation in which MyD88 was absent, then NOD like receptors could compensate the absence. NOD like receptors are intracellular sensors and activate NFkB to initiate the same inflammatory response that the TLRs do. Since MyD88 stimulates signals to TLRS, which then initiate inflammatory responses, NOD like receptors can be used in its place because they also initiate these responses (Murphy, 2012, pp. 90-92).
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