This proposal underline three specific aims.
First,to determine the effect ...
... middle of paper ...
...d male-typical mating behavior.
Another factor that might be involved in the sex difference in testosterone’s effects on neural plasticity and behavior is TrkB(T1), a highly expressed isoform of TrkB, that lacks the kinase binding intracellular domain and absorbs BDNF inhibiting its function (1,2). It is possible that high levels of TrkB (T1) in the female inhibit the actions of testosterone on synaptic plasticity and thereby establishes the sex differences in the brain and behavior. There is precedence for this mechanism in the mammary gland. Dimorphisms in the development of the mammary gland are mediated by androgen actions on TrkB (T1) (5). T1 is present in the male mammary gland due to circulating androgens and absent in the female (5). TrkB (T1) in the male prevents BNDF from attracting axons to the gland, creating a degenerate mammary gland in the males (5).
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