The Association Of Pyrethroid Pesticides And Attention Deficit Hyperactivity Disorder

The Association Of Pyrethroid Pesticides And Attention Deficit Hyperactivity Disorder

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The Association of Pyrethroid Pesticides And Attention Deficit Hyperactivity Disorder
Richardson et. al. Developmental Pesticide Exposure Reproduces Features of Attention Deficit Hyperactivity Disorder. The FASEB Journal 29.5 (2015): 1960-972.
The association of pyrethroid pesticides and attention-deficit/hyperactivity disorder has implications that could affect the way we think about pesticides and health. The fact that there is evidence suggesting a link between these two things may be very important to public health. As a result, precautions and/or changes could definitely be present in the near future. This is not a small finding, and it could be the first of many to suggest a need for further investigation into the use of pesticides. With this evidence, precautions and/or changes need to be made, especially, in children and expecting/nursing mothers
Pyrethroids are a toxin, and they affect every living thing they come in contact with. They are thought to be safer because they are derived from plants, but many plants are also toxic (1). The thought that these will not affect humans in any way is senseless, and already repercussions are being seen. If it is linked to ADHD, it could also be linked to other mental and even physical impairments. These health implications are real and could possibly get worse with the growing use of this pesticide.
Pyrethroid is not the only pesticide to worry about. All pesticides should be investigated. Farmers that use these pesticides have shown to have higher rates of depression and suicide, another reason to show concern when using pesticides (2). ADHD in children is not the only thing to worry about; there could also be a link between adults who commit suicide and their exposure to pest...


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...Methylphenidate, a drug commonly used in patients with ADHD, was given to the deltamethrin exposed mice, and it suspended locomotor activity. A test was conducted over three days to monitor the activity of controlled mice and deltamethrin-exposed mice to see if there were deficits in habituation. It was noted that activity decreased in both the controlled and deltamethrin-exposed mice. However, deltamethrin exposed mice still continued to exhibit increased locomotor activity.
In an attempt to evaluate the role of dopamine receptors in the male offspring who exhibit hyperactivity, apamorphine was administered to mice that had been pretreated to deplete dopamine levels. The apamorphine replenished dopamine levels in all mice,
however activity was increased in the males who were developmentally exposed to deltamethrin, with no increased activity displayed in females.

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