More handpicked essays just for you.
Possible Effects Of Dementia On Health
Alzheimer's effects on society
Effects of Alzheimer's on patient and family
Don’t take our word for it - see why 10 million students trust us with their essay needs.
Recommended: Possible Effects Of Dementia On Health
Even in a world of modern medicine and major medical advancements like the world has never seen before, some diseases still continue to plague the human race and confuse even some of the brightest scientists today. Unfortunately, Alzheimer Disease (AD) is one of them and it affects between 2.4 and 4.5 million people in America. Alzheimer’s is usually diagnosed in people over the age of 65, but in rarer cases people as young as 16 have it. Since it is a degenerative disease, patients develop it with few symptoms at an earlier stage, but then it gradually becomes more predominant in how the patient lives his or her life, developing into dementia ⨥. Alzheimer’s Disease was discovered by a neurologist named Dr. Alois Alzheimer in 1906. Alzheimer was born in Markbreit, Germany he attended a variety of universities including Berlin, Tübingen, and Würzburg, and earned his medical degree in 1887. Alzheimer became research assistant to Emil Kraepelin at the Munich medical school, where he created a new laboratory for brain research. Later, after becoming a professor of psychiatry and neur...
The article “Cracking the Alzheimer’s code” by Linda Marsa discusses the history, discoveries and advancements for Alzheimer’s disease. The discovery of Alzheimer’s disease was revealed through a German physician named Alois Alzheimer. Alzheimer first discovered Alzheimer’s in the year 1901 while he was interviewing a mentally Ill patient named Auguste Dexter. The beginning of his discovery was due to the fact that Dexter was exhibiting uncontrollable behaviors that included jealously, screaming, confusion and paranoia. After Dexter had passed away, Alzheimer saw this as an opportunity to examine her brain under a microscope in thin pieces. To Alzheimer’s surprise, he discovered two abnormal substances on brain slices that were called amyloid
Alzheimer’s disease was first defined in 1906 by a German psychiatrist, Alois Alzheimer. Alzheimer's disease is the most common form of dementia. It is a progressive brain disorder in which the nerve cells in the brain gradually die off. It is estimated that 26 million people world-wide are afflicted by Alzheimer’s and of those, approximately 4.5 million live in the United States. It is said to be the seventh leading cause of death in the USA and the fifth leading cause of death for those over age 65. Seventeen percent of women and ten percent of men age 55 and older can expect to develop Alzheimer’s (apa.org, 2009). Researchers report that this disease is more prevalent in African Americans and Hispanics than in whites (Crandell, Crandell, and Zanden, 2009, p. 578).
Stem cells are a type of unspecialized cells that can revive themselves through the process of cell division (Abdel-Salam and Oma, 2011). They have a phenomenal potential to differentiate into other specific cell types in a living organism. These cells can also be activated to turn into organ-specific cells or tissues with defined activity but only under specific experimental set-up. Stem cells have the ability to remain as they are or divide further to renew deteriorated and damaged tissue cells. Recently it has been discovered that stem cells can be used to treat Alzheimer’s disease but still a lot of clinical trials are being conducted to find a specific government approved treatment. Alzheimer’s disease is the most common cause of dementia. The very first stages of Alzheimer’ include symptoms like confusion and using the right words while talking. As it gets on to later stages symptoms like loss of memory and mood swings start to appear (Begley et al., 2001). Any sort of stem cell treatment is not available to Alzheimer’s patients yet as researchers are still conducting new experiments. There are multiple neurons that get damaged by Alzheimer’s which makes the problem even more complex and different approach needs to be followed every time a clinical trial is conducted.
Alzheimer is a dementia type of disease named after Dr. Alois alzheimer that slowly destroys memory and thinking skills,and eventually , the ability to do simple things, or recognize their family. The first case occurred in the 1906 when a woman died on a unusual mental illness. After she died Dr.Alois examined her brain , amyloid plaques and neurofibrillary. Alzheimer’s is in older people the most common cause of dementia. Dementia is a loss of remembering ,thinking and reasoning skills, that intervenes with your daily life and activities. It is very common in people over sixty years of age. People younger than sixty years can also
The second view advocates the merits of research: AD research cannot be done without AD patients in all the stages of their disease. This is an indubitable fact, because how else can one track the development of the disease (especially the characteristic biological changes)? By invoking the argument of scientific progress and the possibility of finding a cure, the participation of Alzheimer’s disease patients in research is no longer a matter of possibility but of absolute necessity.
performance that involves, but is not limited to, a loss in at least 2 of the
Alzheimer’s disease is a complex illness that affects the brain tissue directly and undergoes gradual memory and behavioral changes which makes it difficult to diagnose. It is known to be the most common form of dementia and is irreversible. Over four million older Americans have Alzheimer’s, and that number is expected to triple in the next twenty years as more people live into their eighties and nineties. (Johnson, 1989). There is still no cure for Alzheimer’s but throughout the past few years a lot of progress has been made.
The first case of dementia was discovered in 1906 by a German physiatrist Alois Alzheimer. It was first observed in a female patient and she was forty-one years old her name was Auguste D. Dr. Alzheimer observed a decline in the patient’s cognitive abilities. She lost her memory, she exhibited behavioral issues, and she suffered from hallucinations, lost the ability to comprehend language, disorientation and lost her speech. After Augusta’s passing Dr. Alzheimer preformed an autopsy that showed the classic triangles and knots we associate today with Alzheimer. Those triangles and knots are a proteins and plaque. The brain is self looked smaller and had distinct characteristics. Still with modern medicine the only way to diagnose a person with Alzheimer is after their death with an autopsy. (THE ALZHEIMER'S PROJECT, 2014).
Alzheimer’s disease got its name from the German doctor, Dr. Alois Alzheimer. In 1906, he noticed that there were abnormal clumps and bundles of fibers i...
Alzheimer's Disease Introduction to Alzheimer's Alzheimer's disease is a progressive, degenerative disease of the brain. It was first described by the German neuropathologist Alois Alzheimer (1864-1915). in 1905. This disease worsens with advancing age, although there is no evidence. that it is caused by the aging process.
Scientists believe that for most people, Alzheimer's results from a combination of genetics, lifestyle and environmental factors that affect the brain over time. Alzheimer's is caused by specific genetic changes that virtually guarantee a person will develop the disease. The causal effect for this disease is still unknown with fingers pointing to plaques and tangles in the brain. Although the causes of Alzheimer's are not yet fully understood, its effect on the brain is clear. Alzheimer's disease damages and kills brain cells. A brain affected by Alzheimer's disease has many fewer cells and many fewer connections among surviving cells than does a healthy brain. As more and more brain cells die, Alzheimer's leads to enormous brain shrinkage. When doctors examined an Alzheimer's brain tissue under the microscope, they saw two types of abnormalities that are considered the cause of the disease. One of these abnormalities is plaques that clump up, a protein called beta-amyloid which damages and destroys brain cells. In patients with Alzheimer’s the plaques created interfere with cell to cell communication. The other abnormality seen is tangles in the brain. Brain cells depend on an internal support and transport system to carry nutrients and other essential materials throughout their long extensions. This system requires the normal structure and functioning of a protein called tau. In an Alzheimer's patient, the threads of tau protein twist into abnormal tangles inside the brain cells, leading to failure of the transport system. (Alzheimer's Association) (National Institutes of Health, 2012)
Alzheimer’s disease, named after Dr. Alois Alzheimer, is a disease that is on the rise in America and the rest of the world. People should learn as much as they want about this disease, because as you age, your chances of becoming an Alzheimer’s Disease, or AD, patient increases. It is estimated that approximately 3 percent of Americans between the ages of 65 and 74 have the illness, and more than half of all people over age 85 have the ailment.
Alzheimer’s disease (AD) is a progressive, terminal, degenerative brain disease. It is the fourth leading cause of death in adults and currently affects over four million people in the United States. This number is expected to increase over the next several years as the baby boomers age, until it reaches fourteen million by the year 2025.
Alzheimer’s disease or AD is an incurable disorder of the brain that results in loss of normal brain structure and function. In an AD brain, normal brain tissue is slowly replaced by structures called plaques and neurofibrillary tangles. The plaques represent a naturally occurring sticky protein called beta amyloid and in an Alzheimer’s brain, sufferer’s tend to accumulate too much of this protein. Neurofibrillary tangles represent collapsed tau proteins which, in a normal brain along with microtubules, form a skeleton that maintains the shape of the nerve cells. In Alzheimer’s disease, the tau proteins break loose from their normal location and form tangles. Without the support of these molecules, nerve cells collapse and die. As normal brain structure is lost with progression of the disease, brain function also degenerates. Patients afflicted with Alzheimer’s disease display a gradual mental decline. Initially, and most apparently, there is a loss of short-term memory. Eventually, as a patient progresses to later stages of the disease, the brain becomes so damaged that patients can no longer communicate or recognize immediate family or even themselves. They have difficulty walking and standing and frequently fall. In the final stages, they lose bladder and bowel control and have difficulty with swallowing, frequently leaving them malnourished and dehydrated. Eventually, they are forced to remain bedridden and, without the help of life-prolonging measures provided in a hospital, die. However, this level of deterioration is severe and may take as long as twenty years. Because of the disease’s slow progress and its usual later start in a person’s life, a victim of AD will usually die first of natural causes. Under the objectives ...
Alzheimer’s disease is rising at a very high rate. “The number of new cases per year is estimated at 360,000 equating to 980 new cases per day or 40 new cases every hour” (Cummings and Cole 1) This evidence shows that an increasing number of people will discover the effects of a cognitive impairment that will most likely be due to Alzheimer’s disease. As people age, their risk of being diagnosed with this disease increases ...