Wolff-Parkinson White Syndrome Wolff-Parkinson White syndrome is a heart condition where there is an extra electrical pathway or circuit in the heart. The condition can lead to episodes of rapid heart also known as tachycardia. Wolff-Parkinson White syndrome, also known as WPW, is present at birth. People of all ages, even infants, can experience the symptoms related to WPW. Episodes of tachycardia often occur when people are in their teens or early twenties. Most of the time, a fast heart beat
adenosine, antiarrhythmic drugs, and Amiodarone may be used to control or prevent a rapid heartbeat” ("Wolff-Parkinson-white syndrome: Medlineplus"). The downside of these medications is that they are very strong and although they may help you fix the WPW syndrome symptoms, they may end up damaging other important organs in the patient’s body as well. In the case of my brother, he happened to suffer WPW syndrome since he was in my mother’s womb. My mother was given Amiodarone, a drug that relaxes the heart
(and rarely) seen in patients with Wolff-Parkinson-White syndrome. It involves conduction of the atrial signal antegrade down the accessory pathway and retrograde up the AV node. Because ventricular depolarization occurs cell to cell, rather than via the normal conduction system, the QRS is wide and mimics monomorphic ventricular tachycardia. This tachycardia is also known as antidromic AVRT.1 A prior ECG with a delta wave typical of Wolff-Parkinson-White syndrome is helpful for this
electrical abnormalities. It is fairly common, especially in children and women, yet unfamiliar to most people who are not diagnosed with it. There are three types of SVT: atrioventricular nodal re-entry tachycardia, atrial tachycardia, and Wolff-Parkinson-White syndrome. All three types have the same symptoms during episodes, which can last from seconds to hours and include palpitations, fatigue, dizziness, etc. However, the three types have different causes, all of which are unknown. To detect this
Question d, Physical Examination rationale for methodology BP Blood pressure reflects the pressure that is exerted on the walls of the arteries. Varying with cardiac cycle, high with systole and low with diastole. A measurement of the pressure of the blood in the arteries. As ventricles contract you have systolic blood pressure and when the ventricles relax you have diastolic blood pressure. Expressed as a ratio of the systolic over diastolic pressure external influences like caffeine, nicotine
Because ocular muscles contain the “highest density of mitochondria per cell of any type of muscle and thus use large amounts of adenosine triphosphate, mitochondrial disease within the ocular system are rather abundant. Symptoms of these ocular syndromes include progressive external ophthalmop... ... middle of paper ... ...G, Yolanda Lillquist, MD, FRCPC. "Mitochondrial Disease Clinical Manifestations: An Overview." British Columbia Medical Journal 53.4 (2011): 183-87. BC Medical Journal. Web
Sports related cardiac deaths are very sudden and most scientists don’t know why they occur, but researchers are conducting many studies to try to determine the exact cause. What happens when an athlete that is in the best of physical health suffers a sudden cardiac death? The sudden death leaves the community, team, and family shocked and devastated and looking for answers. There has been many studies done trying to determine why athletes suffer sudden cardiac arrest. Although sudden cardiac deaths
Last March, an undefeated basketball squad faced a tragic loss, and unfortunately it was not the game. An undefeated season was on the line for the Fennvile Blackhawks, and the whole town was in attendance. The game went into overtime and Wes Leonard was ready for it. Seconds left in overtime, the score was tied again. It was the last play of the game, and Wes Leonard drove to the basket for a lay up, putting them up by two. Time expired and fans rushed the court looking for the hero that made that
Introduction Atrial fibrillation (AF) is a cardiac arrhythmia. It is the most common arrhythmia and it has implications for patients and anaesthetists alike. The anaesthetist must take into consideration the physiological and pharmacological implications of this common arrhythmia. In a healthy individual receiving a general anaesthetic, the anaesthetist must be aware of the causes and treatment of acute onset AF, both intra-operatively and peri-operatively. Patients with AF often develop a decline
grief, and hope, relating to my past experiences. These experiences triggered strong emotional responses which tapped into my inner feelings. Basically, emotions comprise physical arousal, emotional expression and cognitive process (Ciccarelli & White, 2012). Recall of these events provides me an insight into both experiences and reactions for better understanding of own emotions. Anxiety I feel anxious for almost every semester in university. However, this emotion was the strongest for present