Schizophrenia: Effects on the Brain

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Schizophrenia: Effects on the Brain

Schizophrenia is a disease that affects the brain. It alters brain chemistry and brain form to produce the different behavior in those afflicted with the problem. It appears that these alterations change the inherent I-function in each individual. Loss of important functions associated with this piece of the greater nervous system box is found in these people. To understand why this occurs, it is necessary to have some background knowledge of what schizophrenia does to a person's brain. The conclusion of altered I-function can be drawn as this information is presented. The symptoms of the disease also aid in the explanation. Schizophrenia results in varied and abnormal responses compared to those of normal, healthy individuals. The brain of individuals with the disease endures such changes that their basic functions and operations are altered.

Schizophrenia is an internal disease caused by fundamental organizational differences in the brain. It begins to be noticed in a person's late teens through twenties. Signs of it can be seen from early childhood and it is now being hypothesized that it is actually present during adolescence (1). Since this hypothesis extends to fetal development, brain formation in the earliest stages is effected. The alteration of pathways and deviation of neurotransmitters from normal arrangement is believed to be a factor behind the affliction. The brain of these individuals forms differently. Neurotransmitters are believed to misfire altering the chemical balance of the brain causing behavioral problems. There are deficits in cortical and subcortical cognitive processes (5). There are also enlarged ventricles in the brain causing further impairment of ability (3). When the disease is present there are certain symptoms that are easily recognizable. They fall into three categories overt, negative, and positive. The overt symptoms are delusions, hallucinations, alteration of the senses, inability to sort and then respond appropriately to incoming sensations, an altered sense of self, and changes in behavior (2). The negative symptoms are emotional flatness, inability to start or follow through on activities, brief speech lacking content, and lack of pleasure or interest in life (2). All of these include an inhibition on processing information and further imply a differentiation in the normal self. The basic information above provides a solid background to understanding the disease and already possible problems to a person's I-function can be seen.

The afflicted person's lack of knowledge concerning the disease suggests a hindrance to the I-function.

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