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heart disease apa
heart disease apa
effects of heart problems essay
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Heart Disease. Those two words often conjure images of an elderly man clutching his chest in a fit of pain and subsequently succumbing to a heart attack. Those two words describe the leading cause of death in Americans--nearly 25%1. Unfortunately, many specific heart problems fade away and fall under the umbrella of only two words. This research paper will discuss one of those heart problems, aortic dissection, in much more detail, than just two little words.
The aorta is the largest artery in the body, responsible for the transfer of oxygenated blood from the heart to every organ of the body. Understandably, any disruption in the integrity of the aorta could have catastrophic consequences. An aortic dissection occurs when a tear in the inner layer of the aorta develops. As the tear expands, blood will eventually begin to spill out of the tear and continue down the aorta in between the inner layer and outer layer. As the inner and outer layers begin to separate from each other, they develop into what is called a true and false lumen. An aorta can dissect in various ways. Those different types of dissection are then classified as Type A, B and Type I through III. Type A involves the ascending aorta. Type B does not involve the ascending aorta2. Type I involves the entire aorta, Type II involves only the ascending aorta, and Type III originates in the descending aorta and extends distally and does not involve the ascending aorta or arch.2 Dissections that originate in the descending aorta and then progressively dissect proximally are still considered Type III.
There are several different causes of an aortic dissection. Trauma is a major cause of an aortic dissection, specifically blunt trauma, such as a chest hitting a steering ...
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...ers. Numerous signs and symptoms occur with an aortic dissection, making it very difficult to diagnose. Chest x-ray and chest and abdomen CT angiograms are the two most common tests performed to confirm a diagnosis, with CT angiogram being the modality of choice. If the patient survives the initial onset of an aortic dissection long enough surgical repair may be done. Surgical repair is very risky and does not have a high success rate, however, if surgery proves to be successful a patient’s prognosis greatly increases after 30 days. Perhaps the next time one is faced with the familiar imagery of a man grasping his chest and collapsing due to “heart disease,” instead of assuming the man suffered from the usual heart attack, one will remember that there are countless conditions that heart disease encompasses, and that aortic dissection remains one of the most deadly.
An artery is an elastic blood vessel that transports blood away from the heart. There are two main types of arteries: pulmonary arteries and systemic arteries.
...sures (Milewicz, 2005). Acute dissection may be accompanied by all of the classic signs and symptoms similar to that of a heart attack, or it may be clinically silent. In an effort to decrease the mortality rate of patients suffering from Marfans and have a potentially high risk of aortic dissection, current studies are investigating the safety and practicality of endovascular stent graft repair.
...treat aortic dissection. Verapamil and diltazem are used, because of their vasodilatory and negative inotropic effects (Coughlin). A dopamine infusion may be used throughout the case in order to enhance renal perfusion. Heparin is given prior to the cross-clamping of the aorta to reduce the risk of coagulation disorders. Mannitol can be given prior to the cross-clamp to prevent renal failure during resection of an AAA. Furosemide may be used after the cross-clamp is released to improve urinary output (LaMuralgia, Musch).
To stop the blood flow after damage, body uses three ways to maintain hemostasis; vascular spasm, platelet plug formation, and coagulation. Coagulation is an important process to prevent loss of blood when blood vessels are cut or damaged. Blood clot is a plug of platelet reinforce with the mesh of fibrin. However, a person with Disseminated intravascular coagulation, DIC, the blood clots have formed throughout the blood vessels when does not necessary. It leads to organ damages due to blocked blood vessels; furthermore, it leads to life-threatening bleeding due to wasting clotting factors and platelets when they are needed.
aneurysm. Ruptures from this weakening of the lower aorta rank as the 13th leading cause of
If aortic arch is right sided and the patient has pulmonary stenosis physiology, the entire length of the duct can be well visualized in one view because unlike in those patients with vertical ducts, it does not follow a tortuous course.
The Left Descending Artery (LDA) or more affectionately named the “Widow Maker Artery” due to its low survivability rate if blocked, is the vessel in which the deadliest of heart attacks takes place. Due to its location, right in front of the heart, and function, supplying the heart with blood, makes it the hardest to operate on. The Left Descending Artery descends directly from the main portion of the Aorta and, if blocked for too long (due to plaque or surgery), can result in serious side affects for the body because of lack of blood. Like mentioned previously, 735,000 Americans a year suffer from some form of cardiac arrest, depending on who’s research you look at, survivability rates of a widow maker are from 5% to 10%. This blockage is no laughing
Have you ever felt a chest pain, sudden dizziness, weakness, fatigue, etc.? If you answered “Yes” to any of this, it can be Atherosclerosis. On this paper I’m going explain in detail what does Atherosclerosis means. What are the causes, symptoms, treatments? Can we melt the fatty plaques naturally? And more different information in relation to this subject. Atherosclerosis is not an illness that you get from one day to another, actually is an illness that by age, high pressure, the type of your diet, lack of exercise, even people with diabetes would have atherosclerosis with the time. But is not something that cannot be treated, you can go to the doctor for a checkup, change of diet, increase your physical exercise, etc.
An acute myocardial infraction is commonly known as a heart attack. A heart attack is a serious medical emergency that can cause death if not taken serious! “Every year, there are more than 3 million cases in America.” Says Mayo Clinic. Although, a heart attack can happen at any age the majority of the victims are 40 and up. An attack occurs when the blood supply to a part of the heart is damaged or interrupted. Heart attacks are usually caused by obesity, stress, high blood pressure, smoking and many other diseases or poor decision.
Aortic stenosis can range from mild to severe. If untreated, it can become more severe over time.
This assignment will endeavour to demonstrate the map of medicine as followed within the National Health Service for the diagnosis, treatment and follow-up of abdominal aortic aneurysms (AAA). It shall discuss the current AAA screening programme being implemented across the United Kingdom to include the use of Ultrasound and Computed Tomography (CT) as imaging modalities in the demonstration of AAAs. Both the advantages and disadvantages of the modalities used during the diagnosis and treatment of AAA will be shown.
There are different severity levels of an AAA due to the fact that they can range in size from 3 to 20 cm and the bigger they get, the higher the risk of rupture. B2.66. Aneurysms can appear as either saccular or fusiform in shape. A saccular AAA will be sac-like and the fusiform AAA will be more uniform in its shape of dilation. Differential diagnosis for this disease is a thrombus because both present with abnormal flow patterns. Since an AAA will typically have a slow progression and growth rate, unusual features for an AAA are ones that grow progressively fast in a short time period such as months.
Major veins and arteries are located in similar positions deep below skin and muscle tissue, make blood color and rate of bleeding important identifying factors. A few notable arteries and veins are located above the heart and into the neck, with the carotid arteries that pump blood into the brain and the jugular veins that drain blood back to the heart. Several large arteries and veins also flow through the extremities, specifically in the upper arm with brachial arteries and veins and in the upper leg with the femoral artery and vein. These are all connected to large major vessels within the trunk, made up of the upper chest and the upper and lower abdomen. These large vessels connect to those in the extremities and head, as well as circulating through internal organs, and because of this these structures are not only susceptible to external bleeding but internal bleeding as well (786, 811). Internal bleeding is another type of hemorrhage that can occur with hypovolemic shock, and is caused by damage from blunt force brings damage to internal organs or causes blood vessels within the body to rupture, causing blood to escape into the body cavity. Injuries with traumatic force can occur when a victim is in a motor vehicle crash, where force from hitting the steering wheel can cause blunt force to the chest or abdomen or break bones within the body that can damage internal structures. Internal bleeding can also result from an open wound, commonly with penetrative wounds such as with a gunshot or knife stabbing (Red Cross 161). Other notable causes of internal bleeding may be caused by vascular disorders, as with aneurysms (Kolecki “Causes”), that occur when high blood pressure causes arterial wall to weaken and balloon, and possi...
A coronary angiogram, also called coronary angiography, is an X-ray procedure used to look at the arteries in the heart. In this procedure, a dye (contrast dye) is injected through a long, hollow tube (catheter). The catheter is about the diameter of a piece of cooked spaghetti and is inserted through the groin, wrist, or arm. The dye is injected into each artery, and X-rays are then taken to show if there is a blockage in the arteries of the heart.